Presentation is loading. Please wait.

Presentation is loading. Please wait.

Coexpression of other immune genes with ImSig core signatures.

Published byВалерий Розенберг Modified over 5 years ago

Similar presentations

Presentation on theme: "Coexpression of other immune genes with ImSig core signatures."— Presentation transcript:

1 Coexpression of other immune genes with ImSig core signatures.
Coexpression of other immune genes with ImSig core signatures. A, Correlation network of genes associated with the immune clusters during trachomatis infection. ImSig genes are colored according to the different immune cell types they represent, whereas the genes coclustering with the ImSig immune genes are shown as nodes without color and reduced in size. Highlighted with a greater node size and label are a few well-known immune-modulatory genes present in the immediate vicinity of the signature genes. B, Bar plots of the average expression intensity of a few well-known immune-modulatory genes across the three patient groups. Ajit J. Nirmal et al. Cancer Immunol Res 2018;6: ©2018 by American Association for Cancer Research

Download ppt "Coexpression of other immune genes with ImSig core signatures."

Similar presentations

Identification of combination treatment–responsive dysfunctional tumor-infiltrating CD8+ T cell population. Identification of combination treatment–responsive.

Identification of combination treatment–responsive dysfunctional tumor-infiltrating CD8+ T cell population. Identification of combination treatment–responsive.
BCR–ABL1 B-ALLs feature DNA methylation and expression signatures centered aroundIL2RA(CD25). BCR–ABL1 B-ALLs feature DNA methylation and expression signatures.

BCR–ABL1 B-ALLs feature DNA methylation and expression signatures centered aroundIL2RA(CD25). BCR–ABL1 B-ALLs feature DNA methylation and expression signatures.
Correlation of log-transformed signal intensity from two Affymetrix microarray hybridizations using platelet RNA. Plotted are those probesets with an average.

Correlation of log-transformed signal intensity from two Affymetrix microarray hybridizations using platelet RNA. Plotted are those probesets with an average.
The immune metagene model has prognostic value in IBE but not in IBD breast cancer. The immune metagene model has prognostic value in IBE but not in IBD.

The immune metagene model has prognostic value in IBE but not in IBD breast cancer. The immune metagene model has prognostic value in IBE but not in IBD.
Topological overlap matrix (TOM) plots of weighted, gene coexpression networks constructed from one mouse studies (A–F) and four human studies including.

Topological overlap matrix (TOM) plots of weighted, gene coexpression networks constructed from one mouse studies (A–F) and four human studies including.
Representative multiplex immunofluorescence image of synovial sarcoma with increased PD-L1/PD-1/CD8 cell densities at the tumor-invasive margin demonstrating.

Representative multiplex immunofluorescence image of synovial sarcoma with increased PD-L1/PD-1/CD8 cell densities at the tumor-invasive margin demonstrating.
IL-6 is dispensable for the suppressive activity of MDSC on primary CD4+ T-cell activation. IL-6 is dispensable for the suppressive activity of MDSC on.

IL-6 is dispensable for the suppressive activity of MDSC on primary CD4+ T-cell activation. IL-6 is dispensable for the suppressive activity of MDSC on.
Treatment of human MCC tumors with intralesional IFNβ is associated with MHC-I upregulation. Treatment of human MCC tumors with intralesional IFNβ is associated.

Treatment of human MCC tumors with intralesional IFNβ is associated with MHC-I upregulation. Treatment of human MCC tumors with intralesional IFNβ is associated.
B7-H4 expression correlates with MHC-I expression and improved prognosis in patients with breast cancer. B7-H4 expression correlates with MHC-I expression.

B7-H4 expression correlates with MHC-I expression and improved prognosis in patients with breast cancer. B7-H4 expression correlates with MHC-I expression.
Caspase-1 from myeloid cells induces tumor proliferation via MyD88 oncogenic signaling. Caspase-1 from myeloid cells induces tumor proliferation via MyD88.

Caspase-1 from myeloid cells induces tumor proliferation via MyD88 oncogenic signaling. Caspase-1 from myeloid cells induces tumor proliferation via MyD88.
Volume 127, Issue 2, Pages (August 2004)

Volume 127, Issue 2, Pages (August 2004)
Volume 37, Issue 6, Pages (December 2012)

Volume 37, Issue 6, Pages (December 2012)
Chromosome 8 cDNA microarray gene expression profile of the amplified regions of 8p11–12 in SUM-44, -52, and -225 cells versus MCF10A HME control cells.

Chromosome 8 cDNA microarray gene expression profile of the amplified regions of 8p11–12 in SUM-44, -52, and -225 cells versus MCF10A HME control cells.
The identification of leucocyte subset-specific type 1 interferon (IFN-1) modules. The identification of leucocyte subset-specific type 1 interferon (IFN-1)

The identification of leucocyte subset-specific type 1 interferon (IFN-1) modules. The identification of leucocyte subset-specific type 1 interferon (IFN-1)
Quantification of MHC-I, β2m, and T-cell subsets.

Quantification of MHC-I, β2m, and T-cell subsets.
Gene expression changes associated with response to combination CPI-444 and anti–PD-L1 treatment in MC38 tumors. Gene expression changes associated with.

Gene expression changes associated with response to combination CPI-444 and anti–PD-L1 treatment in MC38 tumors. Gene expression changes associated with.
Distinct molecular and clinical correlates of H3F3A mutation subgroups

Distinct molecular and clinical correlates of H3F3A mutation subgroups
Derivation of ImSig. Derivation of ImSig. A, An example of a correlation network generated from a tissue data set where nodes represent unique genes and.

Derivation of ImSig. Derivation of ImSig. A, An example of a correlation network generated from a tissue data set where nodes represent unique genes and.
Correlation of RSPO expression to LGR5 expansion in gastric cancer.

Correlation of RSPO expression to LGR5 expansion in gastric cancer.
Leukemic blasts express a “hypoxia signature

Leukemic blasts express a “hypoxia signature

Similar presentations

Ads by Google