Presentation is loading. Please wait.

Presentation is loading. Please wait.

MR scan of brain fluid-attenuated inversion recovery (FLAIR) (A) and short tau inversion recovery (STIR) (B, C) showing asymmetrical hyperintensities affecting.

Published byEleanore Copeland Modified over 5 years ago

Similar presentations

Presentation on theme: "MR scan of brain fluid-attenuated inversion recovery (FLAIR) (A) and short tau inversion recovery (STIR) (B, C) showing asymmetrical hyperintensities affecting."— Presentation transcript:

1 MR scan of brain fluid-attenuated inversion recovery (FLAIR) (A) and short tau inversion recovery (STIR) (B, C) showing asymmetrical hyperintensities affecting the left nucleocapsular region, thalamus and insula (A), and hyperintense longitudinally extensive lesions in the spinal cord. MR scan of brain fluid-attenuated inversion recovery (FLAIR) (A) and short tau inversion recovery (STIR) (B, C) showing asymmetrical hyperintensities affecting the left nucleocapsular region, thalamus and insula (A), and hyperintense longitudinally extensive lesions in the spinal cord. (Copyright © S Karger AG, Basel; Niemeyer et al36). Sonja Emily Leonhard et al. Pract Neurol 2018;18: ©2018 by BMJ Publishing Group Ltd

Download ppt "MR scan of brain fluid-attenuated inversion recovery (FLAIR) (A) and short tau inversion recovery (STIR) (B, C) showing asymmetrical hyperintensities affecting."

Similar presentations

Neurology Resident and Fellow Section

Neurology Resident and Fellow Section
Nat. Rev. Neurol. doi: /nrneurol

Nat. Rev. Neurol. doi: /nrneurol
Silent Brain Infarcts by Yi-Cheng Zhu, Carole Dufouil, Christophe Tzourio, and Hugues Chabriat Stroke Volume 42(4):1140-1145 March 28, 2011 Copyright ©

Silent Brain Infarcts by Yi-Cheng Zhu, Carole Dufouil, Christophe Tzourio, and Hugues Chabriat Stroke Volume 42(4): March 28, 2011 Copyright ©
MRI and possible differentiating features with nonconventional MRI

MRI and possible differentiating features with nonconventional MRI
Figure 1 Perivenous distribution of multiple sclerosis lesions

Figure 1 Perivenous distribution of multiple sclerosis lesions
Figure 2. MRI features of patients with MS who had antibodies to myelin oligodendrocyte glycoprotein MRI features of patients with MS who had antibodies.

Figure 2. MRI features of patients with MS who had antibodies to myelin oligodendrocyte glycoprotein MRI features of patients with MS who had antibodies.
Figure 4 Paradoxical immune reconstitution inflammatory syndrome

Figure 4 Paradoxical immune reconstitution inflammatory syndrome
Figure 1 Initial brain imaging (A–C) patient 1; (D–F) patient 2; (G–I) patient 3; (J–L) patient 4; and (M) patient 2. Initial brain imaging (A–C) patient.

Figure 1 Initial brain imaging (A–C) patient 1; (D–F) patient 2; (G–I) patient 3; (J–L) patient 4; and (M) patient 2. Initial brain imaging (A–C) patient.
Reversible posterior leukoencephalopathy syndrome and silent cerebral infarcts are associated with severe acute chest syndrome in children with sickle.

Reversible posterior leukoencephalopathy syndrome and silent cerebral infarcts are associated with severe acute chest syndrome in children with sickle.
Figure 3 Brain MRI findings in patients with MOG-Ab Extensive brain lesions with large diameter (A and B), posterior reversible encephalopathy–like lesions.

Figure 3 Brain MRI findings in patients with MOG-Ab Extensive brain lesions with large diameter (A and B), posterior reversible encephalopathy–like lesions.
(A) Diffusion-tensor imaging assessment of white matter damage after traumatic brain injury (TBI). (A) Diffusion-tensor imaging assessment of white matter.

(A) Diffusion-tensor imaging assessment of white matter damage after traumatic brain injury (TBI). (A) Diffusion-tensor imaging assessment of white matter.
Figure 4 Neuromyelitis optica spectrum disorder brain lesions

Figure 4 Neuromyelitis optica spectrum disorder brain lesions
Figure 1 Brain MRI findings in the present case

Figure 1 Brain MRI findings in the present case
A Case of Neuromyelitis Optica Masquerading as Miller Fisher Syndrome

A Case of Neuromyelitis Optica Masquerading as Miller Fisher Syndrome
Figure MRI of anti-MOG-IgG–associated myelitis

Figure MRI of anti-MOG-IgG–associated myelitis
Figure 2 Spinal cord lesions

Figure 2 Spinal cord lesions
Figure 1 Coronal MRI images showing the evolution of white matter abnormality and atrophy of patient 1 Coronal MRI images showing the evolution of white.

Figure 1 Coronal MRI images showing the evolution of white matter abnormality and atrophy of patient 1 Coronal MRI images showing the evolution of white.
Figure Facial photograph during headache attack and brain and upper cervical cord MRI Facial photograph during headache attack and brain and upper cervical.

Figure Facial photograph during headache attack and brain and upper cervical cord MRI Facial photograph during headache attack and brain and upper cervical.
Fluid-attenuated inversion recovery magnetic resonance imaging at the onset of the clinical investigation (A, B) and 2 months later (C, D). Fluid-attenuated.

Fluid-attenuated inversion recovery magnetic resonance imaging at the onset of the clinical investigation (A, B) and 2 months later (C, D). Fluid-attenuated.
Figure 1. Prebiopsy and postbiopsy MRI

Figure 1. Prebiopsy and postbiopsy MRI

Similar presentations

Ads by Google