1. Public Health Policies and Alcohol-related Liver Disease
Meritxell Ventura-Cots, Maria Pilar Ballester-Ferré, Samhita Ravi, Ramon Bataller 
JHEP Reports 
DOI: /j.jhepr
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2. Fig. 1
Lack of early detection of alcohol-related liver disease worldwide. Consecutive patients with a single (A) and double etiology (B) of liver disease seen in Gatroenterology/Liver centers from 19 patients in the 5 continents were included. Percentage of patients seen at advanced vs early stages of liver disease are shown depending on the etiology of the liver disease. *P<.05. (Adapted from Shah et al. with permission)
AIH, autoimmune hepatitis; ALD, alcohol-related liver disease; DILI, drug-induced liver injury; HBV, hepatitis B virus; HCV, hepatitis C virus; HFE, hemochromatosis; NAFLD, non-alcoholic fatty liver disease; PBC, primary biliary cholangitis; PSC, primary sclerosing cholangitis.
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3. Fig. 2
Factors known to influence excessive alcohol intake and alcohol-related liver disease.
The combined effect of different factors (eg. social, environmental and genetic) influence the amount of alcohol intake at the population and individual levels. Susceptibility to develop ALD is in turn influenced by genetic and environmental factors.
Abbreviations: Alcohol-related liver disease, ALD.
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4. Fig. 3
Interactions between different policies, alcohol intake and alcoholic liver disease.
Graphic representation of the combined effect and interactions between different types of public health policies. The represented polices will impact alcohol consumption and subsequently can reduce the burden of ALD.
Abbreviations: Alcohol-related liver disease, ALD. The images are licenced by Creative Commons.
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