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Published byChaz Inscoe Modified over 9 years ago
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Cold Emergencies Prepared by: Steven Jones, EMT-P
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Thermoregulation Homeostasis requires stable temperature ~98.6ºF
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Thermoregulation n Control mechanism Hypothalamus Peripheral thermoreceptors n Balance between heat production, heat loss
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Hypothalamus The main function of the hypothalamus is homeostasis, or maintaining the body's status quo. Factors such as blood pressure, body temperature, fluid and electrolyte balance, and body weight are held to a precise value called the set-point. Although this set-point can migrate over time, from day to day it is remarkably fixed.
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Heat Production n Metabolism n Voluntary large muscle movement n Shivering
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Heat Loss n Conduction n Convection n Radiation n Evaporation Skin Respiratory tract
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Heat Loss > Heat Production Decreased Body Temperature
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Cold Induced Illness n Results from: Decrease in body temperature outside normal range Prolonged efforts to compensate
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Cold-Related Illness n Local cold injury n Generalized cooling
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Localized Cold Injury n Nonfreezing Chilblains Trench foot n Freezing Frostnip Frostbite
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Chilblains n Caused by chronic exposure to damp, nonfreezing ambient temperatures n Painful, inflammatory lesions on skin n Hands, ears, lower legs, feet common sites n Pruritus, burning, paresthesias n Tends to recur n Rewarm, bandage, elevate
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Chilblains
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Trench Foot n Caused by prolonged skin exposure to cool, wet conditions n Skin becomes pale and mottled n Sloughing, gangrene may occur n Clean, warm, dry bandages; elevation
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Trench Foot
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Frostnip/Frostbite n Local freezing of tissue n Commonly affected areas: Toes, feet Hands, fingers Nose Ears
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Frostnip/Frostbite n Risk Factors Poor clothing Poor nutrition Diabetes Decreased tissue perfusion »Tobacco, tight clothing Vasodilation »ETOH, medications
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Frostnip/Frostbite n Pathophysiology: Phase I Exposure to cold Vasoconstriction Decreased blood flow to periphery Ice crystal formation in extracellular space, ischemia Cellular dehydration, hyperosmolarity
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Frostnip/Frostbite n Pathophysiology: Phase I Edema Increased pressure, blood vessel damage Worsened ischemia Destruction of cellular components
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Frostnip/Frostbite n Pathophysiology: Phase II Tissue is rewarmed Blood flow returns Damaged capillaries leak fluid Swelling occurs Sludging of blood, thrombus formation occurs
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Frostnip n Extremity appears pale, discomfort present n No extracellular ice crystal formation n Symptoms resolve on rewarming n Tissue loss does not occur
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Frostnip
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Frostbite Extent of injury frequently cannot be determined until rewarming occurs
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Frostbite n Signs/Symptoms 1st degree »Partial skin freezing; redness, mild edema; lack of blisters 2nd degree »Full thickness freezing; substantial edema, formation of clear blisters
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Frostbite n Signs/Symptoms 3rd degree »Full-thickness skin and subcutaneous freezing; hemorrhagic blisters, skin necrosis, bluish-gray discoloration 4th degree »Full-thickness damage affecting muscles, tendons, bones; little edema, initially mottled or cyanotic, eventually dry, black, mummified
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Frostbite
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n Management: Short transport ABCs Protect affected area »Bandage »Avoid rewarming, thawing Prevent, treat hypothermia »Remove wet or constrictive clothing »Dry patient »Warm IV fluids Minimal analgesics (NSAIDS) for pain
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Frostbite n Management: Long transport Remove patient from cold Remove clothing from affected area Rewarm in water 100 - 105 0 F until flushing/tingling present Dry gently, bandage Treat concurrent hypothermia Analgesia for pain
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Frostbite n Do NOT: Allow refreezing Massage injured part Allow patient to smoke Puncture or drain blebs
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Hypothermia n Core Temp < 95 ºF n May be caused by: Decreased heat production Excess heat loss n Various associated factors Environment (temperature, wet vs. dry) Energy (food, water) Ambulatory ability
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Hypothermia n Risk factors Extremes of age Those outdoors Hypothyroidism Diabetes, hypoglycemia Alcohol, depressant drug abuse Poor nutrition
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Hypothermia n Pathophysiology Immediate vasoconstriction Catecholamine release Increased HR, RR, BP Shivering until »glucose depleted »temperature below 90 o F Shivering stops rapid cooling Eventual in RR, HR, BP Cardiac Arrest < 86 ºF
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Hypothermia n Pathophysiology Decreased oxygen release to tissues Depression of insulin release, effectiveness Hyperglycemia Depression of ADH release Increased urine output, “cold diuresis”
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Hypothermia n Signs/Symptoms Pallor, shivering Ataxic gait Apathy, drowsiness, coma Slowing pulse rate, respirations Cardiac arrest
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Hypothermia Altered LOC + Cool Environment = THINK Hypothermia
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Hypothermia n ECG changes (mostly late) Bradycardia (possibly unresponsive to atropine) Small, absent P wave Abnormal ST segments, T waves J wave (Osborn Wave)
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Hypothermia n J waves
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Hypothermia n Management Mild = core temperature 90-95 o F Moderate = core temperature 86-89 o F Severe = core temperature <86 o F
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Hypothermia n Management: Mild Hypothermia Handle gently Prevent further heat loss Insulate from cold Add heat to head, neck, chest, groin Warm oral fluids after uncontrolled shivering stops
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Hypothermia n Management: Moderate Hypothermia Prevent further heat loss »Remove wet clothing »Cover with blankets Avoid active rewarming IV with NS EKG
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Hypothermia n Management: Severe Hypothermia Secure airway/assist ventilations Do NOT hyperventilate Avoid rough handling Prevent further heat loss »Remove wet clothing »Cover with blankets EKG, IV with NS Internal rewarming only
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Hypothermia n Hypothermic Cardiac Arrest < 86 ºF »Limit 3 shocks »No medications > 86 ºF » drug dosing intervals »Repeat shocks as core temp rises
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Hypothermia n Hypothermic Cardiac Arrest Resuscitate aggressively Little consideration given to terminating resuscitation efforts
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Hypothermia They’re not dead until they’re warm and dead !!
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