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Cardiac Ultrasound in Emergency Medicine
Anthony J. Weekes MD, RDMS Sarah A. Stahmer MD For the SAEM US Interest Group
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Primary Indications Thoraco-abdominal trauma
Pulseless Electrical Activity Unexplained hypotension Suspicion of pericardial effusion/tamponade
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Secondary Indications
Acute Cardiac Ischemia Pericardiocentesis External pacer capture Transvenous pacer placement
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Main Clinical Questions
What is the overall cardiac wall motion? Is there a pericardial effusion?
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Cardiac probe selection
Small round footprint for scan between ribs 2.5 MHz: above average sized patient 3.5 MHz: average sized patient 5.0 MHz: below average sized patient or child
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Main cardiac views Parasternal Subcostal Apical
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Wall Motion Normal Hyperkinetic Akinetic
Dyskinetic: may fail to contract, bulges outward at systole Hypokinetic
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Orientation Subcostal or subxiphoid view
Best all around imaging window Good for identification of: Circumferential pericardial effusion Overall wall motion Easy to obtain – liver is the acoustic window\
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Subcostal View Most practical in trauma setting
Away from airway and neck/chest procedures
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Subcostal View Liver as acoustic window
Alternative to apical 4 chamber view
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Subcostal View
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Subcostal View
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Subcostal View Angle probe right to see IVC
Response of IVC to sniff indicates central venous pressure No collapse Tamponade CHF PE Pneumothorax
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Parasternal Views Next best imaging window Good for imaging LV
Comparing chamber sizes Localized effusions Differentiating pericardial from pleural effusions
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Parasternal Long Axis Near sternum 3rd or 4th left intercostal space
Marker pointed to patient’s right shoulder (or left hip if screen is not reversed for cardiac imaging) Rotate enough to elongate cardiac chambers
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Parasternal Long Axis
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Parasternal Long Axis View
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Parasternal Short Axis
Obtained by 90° clockwise rotation of the probe towards the left shoulder (or right hip) Sweep the beam from the base of the heart to the apex for different cross sectional views
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Parasternal Short Axis View
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Parasternal Short Axis
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Apical View Difficult view to obtain
Allows comparison of ventricular chamber size Good window to assess septal/wall motion abnormalities
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Apical Views Patient in left lateral decubitus position
Probe placed at PMI Probe marker at 6 o’clock (or right shoulder) 4 chamber view
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Apical 4 chamber view Marker pointed to the floor
Similar to parasternal view but apex well visualized Angle beam superiorly for 5 chamber view
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Apical 4 chamber view
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Apical 2 chamber view Patient in left lateral decubitus position
Probe placed at PMI Probe marker at 3 o’clock 2 chamber view
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Apical 2 chamber view Good look at inferior and anterior walls
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Apical 2 chamber view From apical 4, rotate probe 90° counterclockwise
Good view for long view of left sided chambers and mitral valve
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Abnormal findings Pericardial Effusion
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Case Presentation 45 year old male presents with SOB and dizziness for 2 days. He has a long smoking history, and has complained of a non-productive cough for “weeks” Initial VS are BP 88/palp, HR 140 PE: Neck veins are distended Chest: Clear, muffled heart sounds Bedside sonography was performed
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Echo free space around the heart
Pericardial effusion Pleural effusion Epicardial fat (posterior and/or anterior) Less common causes: Aortic aneurysm Pericardial cyst Dilated pulmonary artery
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Size of the Pericardial Effusion
Not Precise Small: confined to posterior space, < 0.5cm Moderate: anterior and posterior, 0.5-2cm (diastole) Large: > 2cm
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Pericardial Fluid: Subcostal
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Clinical features of Pericardial effusion
Pericardial fluid accumulation may be clinically silent Symptoms are due to: mechanical compression of adjacent structures Increased intrapericardial pressure
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Pericardial Effusion:Asymptomatic
Up to 40% of pregnant women Chronic hemodialysis patients one study showed 11% incidence of pericardial effusion AIDS CHF Hypoproteinemic states
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Symptoms of Pericardial Effusion
Chest discomfort (most common) Large effusions: Dyspnea Cough Fatigue Hiccups Hoarseness Nausea and abdominal fullness
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Cardiac Tamponade Increased intracardiac pressures
Limitation of ventricular diastolic filling Reduction of stroke volume and cardiac output
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Ventricular collapse in diastole
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Tamponade Perefftamponade
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Hypotension
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Abnormal findings Is the cause of hypotension cardiac in etiology?
Is it due to a pericardial effusion? Is is due to pump failure?
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Unexplained Hypotension
Cardiogenic shock Poor LV contractility Hypovolemia Hyperdynamic ventricules Right ventricular infarct/large pulmonary embolism Marked RV dilitation/hypokinesis Tamponade RV diastolic collapse
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Cardiogenic shock Dilated left ventricle Hypocontractile walls Chf avi
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Hypovolemia Small chamber filling size Aggressive wall motion
Flat IVC or exaggerated collapse with deep inspiration
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Massive PE or RV infarct
Dilated Right ventricle RV hypokinesis Normal Left ventricle function Stiff IVC
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Case presentation ? overdose
27 yo f brought in with “passing out” after night of heavy drinking. Complaining of inability to breathe! PE: Obese f BP 88/60 HR 123 Ox 78% Chest: clear Ext: No edema Bedside sonography was performed
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PE
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Chest pain then code 55 yo male suffered witnessed Vfib arrest in the ED ALS protocol - restoration of perfusing rhythm Persistant hypotension ED ECHO was performed
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R sided leads
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Non Traumatic Resuscitation
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Direct Visualization Is there effective myocardial contractility?
Asystole Myocardial “twitch” Hypokinesis Normal Is there a pericardial effusion?
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ECHO in PEA Perform ECHO during “quick look” and in pulse checks
Change management based on “positive” findings Pericardial tamponade Pericardiocentesis Hyperdynamic cardiac wall motion Volume resuscitate
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ECHO in PEA RV dilatation Profound hypokinesis Asystole
Hypoxic?? – Likely PE ECG – IMI with RV infarct? Profound hypokinesis Inotropic support Asystole Follow ACLS protocols (for now) Early data suggesting poor prognosis
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ECHO in PEA False positive cardiac motion Transthoracic pacemaker
Positive pressure ventilation
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Case presentation Morbidly obese female with severe asthma
Intubated for respiratory failure Subcutaneous emphysema developed Bilateral chest tubes placed Persistent hypotension at 90/palp Dependent mottling noted ECHO was performed
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Ineffective cardiac contractions
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Optimizing Performance
Assessing capture by transthoracic pacemaker Pericardiocentesis Transvenous pacemaker placement
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Optimizing Performance
Assessment of capture by transthoracic pacemaker Ettin D et al: Using ultrasound to determine external pacer capture JEM 1999
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Case Presentation 70 yo f collapsed in lobby. She was brought into the ED apneic, hypotensive. She was quickly intubated and volume resuscitation begun. VS: BP 80/50 HR 50 Afebrile Physical exam : Thin, minimally responsive f. Clear lungs, nl heart sounds, abdomen slightly distended with decreased bowel sounds. No HSM, ? Pelvic mass ECG: SB, LVH, no active ischemia
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Clinical questions? Why is she hypotensive? Volume loss Pump failure
?Ruptured AAA Pump failure Bedside sonography was performed while we were waiting for the “labs”
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Increase HR with PM “on”
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What did this tell us? Normal wall motion
No pericardial/pleural effusion Good capture with the transthoracic PM
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Asystole w/ Transthoracic PM
Asystole epm.avi
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Optimizing performance
Pericardiocentesis Standard of care by cardiology/CT surgery to use ECHO to guide aspiration
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US Guided- Pericardiocentesis
Subcostal approach Traditional approach Blind Increased risk of injury to liver, heart Echo guided Left parasternal preferred for needle entry or… Largest area of fluid collection adjacent to the chest wall
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Large pericardial effusion
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Technique
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Optimizing performance
Placement of transvenous pacemaker Aguilera P et al: Emergency transvenous cardiac pacing placement using ultrasound guidance. Ann Emerg Med 2000
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Untimely end 30 yo brought in after he “fell out”
Ashen m with no spontaneous respirations VS: No pulse, agonal rhythm on monitor Intubated/CPR Transvenous pacemaker placed, no capture. ECHO showed
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PM placement
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Penetrating Chest Trauma
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Penetrating Cardiac Trauma
Physician’s ability to determine whether there is a hemodynamically significant effusion is poor Beck’s Triad Dependent on patient cardiovascular status Findings are often late Determinants of hemodynamic compromise Size of the effusion Rate of formation
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Penetrating Cardiac Injury
Emergency department echocardiography improves outcome in penetrating cardiac injury. Plummer D et al. Ann Emerg Med. 1992 28 had ED echo c/w 21 without ED echo Survival: 100% in echo, 57.1% in nonecho Time to Dx: 15 min echo, 42 min nonecho
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Penetrating Cardiac Injury
The role of ultrasound in patients with possible penetrating cardiac wounds: a prospective multicenter study. Rozycki GS: J Trauma. 1999 Pericardial scans performed in 261 patients Sensitivity 100%, specificity 96.9% PPV: 81% NPV:100% Time interval BUS to OR: /- 5.9 min
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Penetrating Cardiac Trauma
Emergency Department Echocardiography Improves Outcome in Penetrating Cardiac Injury Plummer D, et al. Ann Emerg Med 21: , 1992. “Since the introduction of immediate ED two-dimensional echocardiography, the time to diagnosis of penetrating cardiac injury has decreased and both the survival rate and neurologic outcome of survivors has improved.”
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Stab wound to the chest SAEM Pericardial effusion
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Penetrating Cardiac Trauma
Echocardiographic signs of rising intrapericardial pressure Collapse of RV free walls Dilated IVC and hepatic veins Goal: Early detection of pericardial effusion Develops suddenly or discretely May exist before clinical signs develop Salvage rates better if detected before hypotension develops
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Technical Problems Subcutaneous air Pneumopericardium
Mechanical ventilation Scanning limited by: Pain/tenderness Spinal immobilization Ongoing procedures
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Technical Problems Narrow intercostal spaces Obesity Muscular chest
COPD Calcified rib cartilages Abdominal distention
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Sonographic Pitfalls Pericardial versus pleural fluid Pericardial clot
Pericardial fat
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Pericardial or Pleural Fluid
Left parasternal long axis: Pericardial fluid does not extend posterior to descending aorta or left atrium Subcostal: No pleural reflection between liver and R sided chambers A pleural effusion will not extend between to RV free wall and the liver
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Pleural and Pericardial fluid
Pleural perica
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Pleural effusion
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Blunt Cardiac Trauma Cardiac contusion Cardiac rupture
Valvular disruption Aortic disruption/dissection
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Blunt Cardiac Trauma Pericardial effusion
Assess for wall motion abnormality RV dyskinesis (takes the first hit) Assess thoracic aorta: Hematoma Intimal flap Abnormal contour Valvular dysfunction or septal rupture
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Cardiac Contusion Akinetic anterior RV wall Small pericardial effusion
Diminished ejection fraction
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RV Contusion
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Blunt Cardiac Trauma Assess thoracic aorta
Hematoma Intimal flap Abnormal contour Requires TEE and expertise! Valvular dysfunction or septal rupture Requires expertise beyond our scope
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Summary Bedside ECHO can help assess:
Overall cardiac wall motion Identify clinically significant pericardial effusions Useful in the assessment of the patient with: Unexplained hypotension Dyspnea Thoracic trauma
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