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Acute Compartment Syndrome
Marc Hirner
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Demographics Incidence: 69% due to trauma Men 7.3/100,000
Women 0.7/100,000 69% due to trauma 36% fx tibia 9.8% distal radius 23% soft tissue injury without fx 10% on anticoagulants
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Case 1 Patient with ? Trivial knee injury Seen in ED and admitted Registrar to ward , pulseless limb Was in fact a knee dislocation that reduced spontaneously End result popliteal artery repair , fasciotomy , ligament reconstruction and eventual BKA
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Case 2 Simple fibula fracture Referred to White Cross several days after injury with tight swollen calf Diagnosed acute compartment syndrome 5 days late Fasciotomy of no use as muscles necrotic
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Case 3 Child required IV access so the tibia was used for rapid infusion Fluid into the calf Acute compartment syndrome Orthopaedics notified late Fasciotomy no use as muscles necrotic
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Etiology
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Pathophysiology Increased compartment pressure
Increased venous pressure Decrease A-V gradient resulting in muscle and nerve ischemia.
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Diagnosis History Clinical exam: the Ps Compartment pressures
Laboratory tests CPK Urine myoglobin
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Clinical Diagnosis The six ‘Ps’: Pressure Pain Paresthesia Paralysis
Pallor Pulselessness
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Pressure Early finding Only objective finding
Refers to palpation of compartment and its tension or firmness
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Pain Out of portion to injury Exaggerated with passive stretch
Earliest symptom but inconsistent Not available in obtunded patient
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Paresthesia Early sign Difficult to interpret
Peripheral nerve tissue is more sensitive than muscle to ischemia Permanent damage may occur in 75 minutes Difficult to interpret Will progress to anesthesia if pressure not relieved
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Paralysis Very late finding Paresis may be present early
Irreversible nerve and muscle damage present Paresis may be present early Difficult to evaluate because of pain
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Pallor & Pulselessness
Rarely present Indicates direct damage to vessels rather than compartment syndrome Vascular injury more of contributing factor to syndrome rather than result
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Compartment Pressure When Technique Confirm clinical exam
Obtunded patient with tight compartments Regional anesthetic Vascular injury Technique Whiteside infusion Stic technique: side port needle Wick catheter Slit catheter
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Stryker Stic System Easy to use Can check multiple compartments
Different areas in one compartment
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Distance From Fracture Effects Pressure
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What is Critical Pressure?
>30 mm Hg as absolute number (Roraback)
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Treatment Lower leg to level of the heart Remove cast
Split all dressings down to skin
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Treatment If concerned refer these patients early
Fasciotomy if continued clinical findings and/or elevated compartment pressure
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Treatment
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Wound Care Soft tissue coverage by 5-7 days Delayed closure
Vascular loop ‘lace technique’ Split thickness skin graft Flaps or free tissue transfer
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NO ONE EVER BLAMES US FOR DOING A FASCIOTOMY BUT MISSING COMPARTMENT SYDROME IS A DISASTER
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