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Figure 8.1 Forms of short-term synaptic plasticity
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Figure 8.1 Forms of short-term synaptic plasticity (Part 1)
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Figure 8.1 Forms of short-term synaptic plasticity (Part 2)
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Figure 8.1 Forms of short-term synaptic plasticity (Part 3)
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Figure 8.1 Forms of short-term synaptic plasticity (Part 4)
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Figure 8.1 Forms of short-term synaptic plasticity (Part 5)
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Figure 8.2 Short-term plasticity at the neuromuscular synapse
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Figure 8.2 Short-term plasticity at the neuromuscular synapse (Part 1)
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Figure 8.2 Short-term plasticity at the neuromuscular synapse (Part 2)
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Figure 8.3 Short-term sensitization of the Aplysia gill withdrawal reflex
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Figure 8.3 Short-term sensitization of the Aplysia gill withdrawal reflex (Part 1)
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Figure 8.3 Short-term sensitization of the Aplysia gill withdrawal reflex (Part 2)
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Figure 8.3 Short-term sensitization of the Aplysia gill withdrawal reflex (Part 3)
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Figure 8.4 Synaptic mechanisms underlying short-term sensitization
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Figure 8.4 Synaptic mechanisms underlying short-term sensitization (Part 1)
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Figure 8.4 Synaptic mechanisms underlying short-term sensitization (Part 2)
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Figure 8.4 Synaptic mechanisms underlying short-term sensitization (Part 3)
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Figure 8.5 Mechanism of presynaptic enhancement underlying behavioral sensitization
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Figure 8.5 Mechanism of presynaptic enhancement underlying behavioral sensitization (Part 1)
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Figure 8.5 Mechanism of presynaptic enhancement underlying behavioral sensitization (Part 2)
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Box 8A Genetics of Learning and Memory in the Fruit Fly
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Figure 8.6 The rodent hippocampus
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Figure 8.7 Long-term potentiation of Schaffer collateral-CA1 synapses
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Figure 8.7 Long-term potentiation of Schaffer collateral-CA1 synapses (Part 1)
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Figure 8.7 Long-term potentiation of Schaffer collateral-CA1 synapses (Part 2)
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Figure 8.7 Long-term potentiation of Schaffer collateral-CA1 synapses (Part 3)
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Figure 8.7 Long-term potentiation of Schaffer collateral-CA1 synapses (Part 4)
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Figure 8.8 Pairing presynaptic and postsynaptic activity causes LTP
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Figure 8.9 LTP at a CA1 pyramidal neuron receiving inputs from two independent pathways
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Figure 8.10 The NMDA receptor channel can open only during depolarization
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Figure 8.11 Signaling mechanisms underlying LTP
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Figure 8.12 Addition of postsynaptic AMPA receptors during LTP
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Figure 8.12 Addition of postsynaptic AMPA receptors during LTP (Part 1)
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Figure 8.12 Addition of postsynaptic AMPA receptors during LTP (Part 2)
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Figure 8.12 Addition of postsynaptic AMPA receptors during LTP (Part 3)
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Box 8B Silent Synapses
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Box 8B Silent Synapses (Part 1)
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Box 8B Silent Synapses (Part 2)
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Figure 8.13 Role of protein synthesis for maintaining LTP
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Figure 8.14 Mechanisms responsible for changes in synaptic transmission during LTP
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Figure 8.14 Mechanisms responsible for changes in synaptic transmission during LTP (Part 1)
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Figure 8.14 Mechanisms responsible for changes in synaptic transmission during LTP (Part 2)
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Figure 8.15 Long-term synaptic depression in the hippocampus
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Figure 8.15 Long-term synaptic depression in the hippocampus (Part 1)
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Figure 8.15 Long-term synaptic depression in the hippocampus (Part 2)
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Figure 8.16 Long-term synaptic depression in the cerebellum
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Figure 8.16 Long-term synaptic depression in the cerebellum (Part 1)
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Figure 8.16 Long-term synaptic depression in the cerebellum (Part 2)
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Figure 8.16 Long-term synaptic depression in the cerebellum (Part 3)
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Figure 8.16 Long-term synaptic depression in the cerebellum (Part 4)
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Figure 8.17 Spike timing-dependent synaptic plasticity in cultured hippocampal neurons
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Figure 8.17 Spike timing-dependent synaptic plasticity in cultured hippocampal neurons (Part 1)
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Figure 8.17 Spike timing-dependent synaptic plasticity in cultured hippocampal neurons (Part 2)
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Figure 8.17 Spike timing-dependent synaptic plasticity in cultured hippocampal neurons (Part 3)
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Box 8C Epilepsy: The Effect of Pathological Activity on Neural Circuitry
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