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ECG Interpretation Criteria Review
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Axis Deviation RAD = If R wave in III > R wave in II
Left Right RAD = If R wave in III > R wave in II LAD = If R wave in aVL > I; and deep S wave in III
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Axis Deviation Criteria
LEAD I LEAD II (or Lead aVF or III) Normal Positive LAD Negative RAD Intermediate axis
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Axis Deviation LAD = possible left anterior fasicular block
RAD = possible left posterior fasicular block
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Right Atrial Abnormality Criteria
Tall P waves in lead II (or III, aVF and sometimes V1)
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Left Atrial Abnormality
Lead II (and I) show wide P waves (second hump due to delayed depolarization of the left atrium) (P mitrale: mitral valve disease) V1 may show a bi-phasic P wave 1 box wide, 1 box deep (biphasic since right atria is anterior to the left atria)
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Right Ventricular Hypertrophy Criteria
In V1, R wave is greater than the S wave - or - R in V1 greater than 7 mm Right axis deviation In V1, T wave inversion (reason unknown)
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Left Ventricular Hypertrophy Criteria
If S wave in V1 or V2 + R wave in V5 or V6 ≥ 35 mm... ...or, R wave > 11 (or 13) mm in aVL or I... ...or, R in I + S in III > 25 mm. Also LVH is more likely with a “strain pattern” or ST segment changes Left axis deviation Left atrial abnormality
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Right Bundle Branch Block Criteria
V1 or V2 = rSR’ - “M” or rabbit ear shape V5 or V6 = qRS Large R waves Right chest leads: T wave inversion (“secondary changes” since they reflect a delay in depolarization not an actual change in depolarization). Complete RBBB: QRS > 0.12 sec. Incomplete RBBB: QRS = 0.10 to 0.12 sec.
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Left Bundle Branch Block Criteria
Wide QRS complex V1 = QS (or rS) and may have a “W” shape to it. V6 = R or notched R and may show a “M” shape or rabbit ears Secondary T wave inversion Secondary if in lead with tall R waves Primary if in right precordial leads
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Incomplete Bundle Branch Blocks
RBBB or LBBB where QRS is between .10 and .12 with same QRS features
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Left Anterior Fascicular Block
Limb leads QRS less width less than 0.12 sec. QRS axis = Left axis deviation (-45° or more) if S wave in aVF is greater than R wave in lead I small Q wave in lead I, aVL, or V6
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Left Posterior Fascicular Block
Right axis deviation (QRS axis +120° or more) S wave in lead I and a Q wave in lead III (S1Q3) Rare
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Bifascicular Block Two of the three fascicles are blocked.
Most common is RBBB with left anterior fascicular block.
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Subendocardial Ischemia Partial occlusion Transmural Infarction (MI) Complete occlusion
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A. Normal ECG prior to MI B. Hyperacute T wave changes - increased T wave amplitude and width; may also see ST elevation C. Marked ST elevation with hyperacute T wave changes (transmural injury) D. Pathologic Q waves, less ST elevation, terminal T wave inversion (necrosis) E. Pathologic Q waves, T wave inversion (necrosis and fibrosis) F. Pathologic Q waves, upright T waves (fibrosis)
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Infarction
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Anterior Infarctions Abnormal Q waves in chest leads
Anterior MI can show loss of R wave progression in the chest leads
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Inferior Infarctions Abnormal Q waves in leads II, III, and aVF
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Lateral Lateral - V5 and V6
High lateral when ST elevation and Q waves localized to leads I and aVL
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Posterior MI Tall R waves in V1,V2 R/S ratio > 1 in V1, V2
The tall, anterior R waves are mirror images of a pathological, posterior Q waves. Absences of right axis deviation (found with RVH) ST segment depression in V1-V3 Often seen with inferior MI
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Infarctions or BBB RBBB & LBBB
T wave inversion and ST segment depression in V1 & V2 (RBBB) and V5 & V6 (LBBB) MI T wave inversion and ST segment depression in additional leads Likely loss of R wave progression
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Infarctions and BBB RBBB and MI
usual ECG changes in leads other than V1 and V2 septal MI - upright T waves in V1 and V2 with just RBBB the T waves should be inverted so upright T waves w/ RBBB are “abnormal” and indicated septal MI
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Infarctions and LBBB Infarctions often damage the left bundle branch leading to a new or recent LBBB expect to see upright T waves in left chest leads septal MI are very difficult to assess with LBBB
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Subendocardial Ischemia
ST Segment depression Anterior leads (I, aVl and V1-V6) Inferior leads (II, III, and aVf) may see ST segment elevation in aVr T wave inversion Poor R wave progression
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Subendocardial Infarction
No Q waves (non-Q wave infarction) Persistent ST segment depression T wave inversion
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Sinus Bradycardia HR less than 60 bpm
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Sinus Tachycardia HR > 100 bpm
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Premature Atrial Complexes (PAC)
Normal conduction Conducted with aberration a fascicles or bundle branch is refractory wide QRS Non-conducted the AV node was still refractory; P wave will be close to the T wave no QRS complex
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Atrial Tachycardia
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AV Nodal Reentrant Tachycardia
Figure 14-6 Rapid recirculating impluse in the AV node area ( beats/min) No P waves (hidden in QRS complex) or may be just before or after the QRS complex Negative P waves in lead II
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Atrial Flutter Sawtooth; F waves (easiest seen in II, III, & aVF)
Atrial rate of about 300 bpm Ventricular rate150, 100 or 75 beats/min 2:1, 3:1 and 4:1
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Atrial Fibrillation No organized depolarization in atria.
Irregular “f waves” can range from looking almost like P waves to a flat line. Atrial rate is about 600 bpm Normal QRS w/ ventricular rate ~ but random & irregular
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Junctional Rhythm
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Accelerated Junctional Rhythm
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WPW
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First Degree AV Block
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2nd Degree AV Block, Type 1
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2nd Degree AV Block, Type 2
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2rd Degree AV Block
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Premature Ventricular Contractions
Characteristics Premature and occur before the next normal beat Wide (> 0.12 ms) and the T wave is usually opposite of the QRS Bizarre looking PVCs usually precede a P wave. A nonsinus P wave may follow the PVC
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PVC Unifocal (monomorphic) PVCs same appearance in the same lead
small focus normal and diseased hearts
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PVC Polymorphic (multifocal and multiform) PVCs
different appearance in the same lead multiform = different coupling intervals multifocal = same coupling intervals usually diseased hearts Multiform
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Idioventricular Rhythm
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Couplet
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Triplet
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Bigeminy and Trigeminy
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Ventricular Tachycardia
...more than three PVCs
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Torsades de Pointes
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Ventricular Fibrillation
Note the course and fine waves
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