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Cardiology II Arrhythmias
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Objectives O Describe the normal conduction in the heart O Describe pathophysiology of bradycardias O Describe pathophysiology of tachycardias O Describe treatment of the above
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Case O A 55 year old male calls 911 because his “heart is racing.” He initially is alert, oriented, and has mild shortness of breath. On physical exam, he has a regular tachycardia at 180, and monitor shows a regular, narrow-complex tachycardia. He denies chest pain. Midway through transport, he becomes less responsive, and his blood pressure drops as he starts sweating profusely.
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Normal Conduction
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ECG waveform
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Normal ECG
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Sinus Arrhythmia
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Bradycardias O Caused by damage to the conduction system resulting in a “broken road” or by drugs (beta blocker, calcium channel blockers) O If due to drugs, reversible. If not, many require a permanent pacemaker O Our goal is to temporarily support the heart rate until one of those two happen. O Atropine or External Pacing are options.
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Sinus Bradycardia
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First Degree AV Block
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Second Degree (Type I)
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Second Degree (Type II)
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Third Degree Block
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Junctional Rhythm
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Accelerated Idioventricular
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Ventricular Escape
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Ventricular fibrillation
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What is this?
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Asystole
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Branch Blocks O “Hiccups” on the conduction highway past the AV node O Can involve the right bundle, the left bundle, or divisions of the left bundle
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Right Bundle Branch Block
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Left Bundle Branch Block
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Bifasicular Block RB/LA
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Bifasicular Block – RB/LP
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Trifasicular Block
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Tachycardias O Any heart rate greater than 100. O The key is using the pattern to determine what is causing the tachycardia O The treatment is even more diverse, thus, knowing the pattern is critical to choosing the correct therapy. O The three main branches of tachycardia are atrial, nodal, and ventricular
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Sinus Tachycardia
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Atrial Fibrillation
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Atrial Fib with Abberancy
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Multifocal Atrial Tachycardia
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Atrial Flutter
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AV Nodal Reentrant Tachycardia
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Wolfe Parkinson White
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Lown-Ganong-Lavine
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AV Reciprocating Tachycardia
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Ventricular Tachycardia
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Torsades de Pointe
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Miscellaneous
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LVH
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RVH
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Hyperkalemia
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Digoxin Toxicity
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Brugada Syndrome
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Wellen’s Syndrome
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Treatment Summary O EMT O ABCs O Call for ALS O Rapid Transport O AEMT O IV O Cardioversion of V-tach, V-fib O Paramedic O Atropine, pacing for bradycardias O Vagal Maneuvers, Adenosine for narrow complex tachyarrhythmias O Amio, cardioversion for wide complex tachyarrhythmia
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