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The CHEST Emergency Medical Conditions MI Zucker, MD
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A dr Z Lecture
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The WHAT: Normal chest films Abnormal patterns Atelectasis Infection Obstructive airway diseases Heart failure Noncardiac edema Pulmonary embolism Aortic dissection …and a few others
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The WHY: Give you a basic approach to acute medical diseases emphasizing CHEST Radiographs Show you the most COMMON diseases Show you commonly MISSED findings
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The NORMAL Chest PA/lateral Portable AP
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The PA (adult and kid) and Lateral Check list Commonly overlooked areas
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The PORTABLE AP The “bottom feeder” of chest radiology
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Pitfalls in Chest Radiology Phase of respiration Position Comparison films Portables
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Poor Inspiration
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The PATTERNS Too dense Too lucent
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Location, Location, Location Lung? Chest wall? Mediastinum? Pleura?
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Lung: Too DENSE Alveolar pattern Interstitial pattern Masses
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Lung: Alveolar Pattern Something of unit/soft tissue/water density replaces the air in the alveolar ducts, alveolar sacs and the alveoli
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Alveolar Lung Pattern: causes PUS WATER BLOOD Lymphoma BAC Alveolar proteinosis
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Alveolar Lung Pattern: findings Increased density Confluence Ill defined margins Air bronchograms
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Lung: Interstitial Pattern Something thickens the interstitium of the lung parenchyma
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What? Edema Inflammatory cells RBC’s Malignant cells Fibrosis
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How? All of them ADD tissue to the peripheral and axial interstitium of the secondary pulmonary lobule
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Secondary Pulmonary Lobules
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Who? Many, many diseases present with the same interstitial patterns You need history, lab, and frequently biopsy to make a specific diagnosis
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A Memory Aid “I Munch Ice Chips In Places Called Igloos”
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Interstitial diseases Idiopathic Malignancy Infection UIP DIP LIP BOOP LAM PEG sarcoid Metastases, lymphoma Viral, PCP, mycoplasma. Fungi, TB, MAC
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Interstitial diseases Congenital Iatrogenic Pulmonary edema NF TS CF Drugs, radiation Cardiogenic, renal, noncardiogenic
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Interstitial diseases Collagen-vascular Inhalational RA, SLE, scleroderma, AS Allergic alveolitis, noxious gases, pneumoconiosis
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The Interstitial Patterns Lines: fine, medium, or coarse Nodules: tiny to 3 cm Reticular: network of crossing lines Reticular-nodular: lines and nodules
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Lines
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Nodules
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Reticular pattern
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Lines and nodules
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What do they mean? Coarse lines mean fibrosis, also called “honeycomb” pattern The other patterns usually mean more active disease, but aren’t specific
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Kerley lines, A and B Thickened secondary lobule septae Often, but not always due to CHF Basically, they are just a slightly specialized intertstitial linear pattern A and B differ only by location
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INFECTION The pneumonias
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BACTERIAL PNEUMONIAS Pyogenic
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The Silhouette Sign If two adjacent structures have the same density, the border between them is not visible. Replace air with an alveolar process and the border between the involved lung and the heart, or diaphragm, or aorta disappears
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The Spine Sign On the lateral view the spine normally progressively looks darker caudally. If it looks whiter, there is an alveolar process in one of the lower lobes.
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Atypical Pneumonias Mycoplasma Chlamydia
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Patterns Diffuse bilateral patchy opacities Diffuse interstitial linear opacities
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Viral Pneumonias Air-trapping Mucus plugs and atelectasis Diffuse interstitial linear and nodular opacities Findings more pronounced in kids
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Pneumocystis carnii Pneumonia Interstitial linear nodular pattern, usually bilateral Followed by diffuse alveolar pattern Early, 10% of CXR’s in PCP can be negative. Later, atypical patterns are fairly common.
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PCP
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Tuberculosis Primary Post-primary
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TB: primary
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TB: postprimary, early
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TB: postprimary, cavitary
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A few more Infections Lung abscess Empyema Fungus
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Lung abscess
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Empyema: Hydro-pneumothorax
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Coccidioidomycosis
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ATELECTASIS Loss of Lung Volume
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Atelectasis: types Obstructive Passive Compressive Cicatricial Adhesive
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Atelectasis: signs Increased density Shift of fissure Elevation of diaphragm Shift of mediastinum Shift of heart Shift of hilum Compensatory hyperinflation
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Subsegmental
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Right upper lobe
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Lower lobes
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Right middle lobe
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Left upper lobe
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ATX: entire lung
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Edema Cardiogenic Renal Noncardiogenic
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Edema: pathogenesis Cardiogenic: increased hydrostatic pressure Noncardiogenic: increased alveolar- capillary membrane permeability Renal: multiple factors
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Cardiogenic Congestive heart failure
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CHF Cephalization 12 wedge pressure Interstitial edema 20 Alveolar edema 25 Cardiomegaly, pleural effusions
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CHF: cephalization
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CHF: interstitial edema
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CHF: alveolar edema
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Renal related Fluid overload Increased permeability CHF
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Edema: renal
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Noncardiogenic edema Near drowning High altitude Drugs Inhalation Hypoxia (ARDS)
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Noncardiogenic edema
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Obstructive lung disease Asthma COPD
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Asthma Hyperinflation Mucus plugs/atelectasis Interstitial inflammation Barotrauma
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Asthma: kid
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Asthma: adult
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COPD Hyperinflation Flat diaphragm Increased retrosternum air space Pulmonary arterial hypertension Look for pneumonia as cause of exacerbation
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COPD
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Pulmonary embolism
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PE: diagnosis Clinical: dyspnea, chest pain, increased RR & PR D-dimer Doppler ultrasound *CXR *CTPA Lung scan Pulmonary angiography
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Chest film Subsegmental atelectasis Small pleural effusion Elevated diaphragm Westermark’s (rare) Hampton’s (rare)
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PE: CXR
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PE: CTPA
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Aortic Dissection
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HYPERTENSION Marfans Coarctation Turners, SLE, pregnancy
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Aortic Dissection Type A: more common, ascending aorta, surgery Type B: descending aorta, trial of medical management
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AD: imaging CXR CTA MRI TEE Angiography
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AD: CXR Mediastinum contour abnormality: abnormal shape or width A change in contour from previous film
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AD: CXR Sensitivity: 80%
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AD: CXR
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AD: CTA axial
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AD: CTA reformat
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…and a few more
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Sickle Cell Disease Cystic Fibrosis
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Sickle Cell Disease
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Cystic Fibrosis
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Goodbye Copyright 2004 MI Zucker, MD
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