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Published byShana Hall Modified over 9 years ago
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Spinal Trauma
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Types Cervical 40% Thoracic 10% Lumbar 3% Dorso lumbar 35% Combination of areas 14%
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Anatomy Spinal cord ends below lower border of L1 Cauda equina is below L1 Mechanical injury - early ischaemia, cord edema - cord necrosis Neurological recovery unpredictable in cauda equina ie. peripheral nerves
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Cauda equine compression
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Cervical spine anatomy Anterior column - Anterior longitudinal ligament+ Anterior annular ligament and anterior half of VB. Middle column – Posterior long. Lig. + Posterior annular ligament +Posterior half of VB. Posterior Column – Lig flavum + superior & Interspinous lig + intertransverse capsular lig + neural arch + pedicle & spinous process.
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Significance Unstable if middle column + either Anterior or Posterior column is damaged Rupture of interspinous ligament is : - associated with avulsion of spinous process - Unstable spine - Further flexion increases neurological injury
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Level of Spinal injury Neurological level is at the most lowest segment with normal motor & sensory function Difficult to determine : - as most muscle efferents receive fibres from more than one level - Closed cord lesions may extend over several cms. - Dermatomes have imprecise boundaries.
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Cord level C2 – C7 = add +1 for cord level T1 – T6 = add +2 T7 – T9 = add +3 T10 = L1, L2 level T11 = L3, L4 level L1 = sacro coccygeal segments
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Degrees of injury Complete - flaccid paralysis + total loss of sensory & motor functions Incomplete - mixed loss - Anterior sc syndrome - Posterior sc syndrome - Central cord syndrome - Brown sequard’s syndrome - Cauda equina syndrome
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Anterior spinal cord syndrome Flexion rotational force to spine Due to compression fracture of vertebral body or anterior dislocation Anterior spinal artery compression Loss of power, reduced pain and temperature below the lesion.
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Posterior cord syndrome Hyperextension injuries Posterior vertebral body fracture Loss of proprioception and vibration sense Severe ataxia
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Central cord syndrome Older age with cervical spondylosis Hyperextension with minor trauma Cord is compressed by osteophytes from vertebral body against thick ligamentum flavum. Damages the central cervical tract UMN lesion to legs (spastic) LMN to arms (flaccid paralysis)
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Brown sequards syndrome Hemisection of the cord Stab injury and lateral mass fractures Uninjured side has good power but absent pinprick and temperature. Spinothalamic tracts cross to opposite side of the cord three segments below.
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Types of bony injury Flexion Extension Flexion with rotation Compression
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Pathophysiology Primary Neurological damage Direct trauma, haematoma & SCIWORA ( Spinal Cord Injury w/o Radiologic Abnormality ) < 8yrs old In 4hrs - Infarction of white matter occurs In 8hrs - Infarction of grey matter and irreversible paralysis Secondary damage Hypoxia Hypoperfusion Neurogenic shock Spinal shock
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Hypoxia Lesions above C 5 – damage to diaphragm leads to 20% reduction in vital capacity Rx Phrenic n. pacing Lesions at D 4-6 – reduces vital capacity if < 500ml patient is ventilated Intercostal nerve paralysis Atelectasis – poor cough V/Q mismatch Reduced compliance of lung – muscle fatigue.
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Neurogenic shock Lesions above D6 Minutes – hours (fall of catecholamines may take 24 hrs) Disruption of sympathetic outflow from D1 - L2 Unapposed vagal tone Peripheral vasodilatation Hypotension, Bradycardia & Hypothermia BUT consider haemmorhagic shock if – injury below D6, other major injuries, hypotension with spinal fracture alone without neurological injury.
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Spinal shock Transient physiological reflex depression of cord function – ‘concussion of spinal cord’ Loss anal tone, reflexes, autonomic control within 24-72hr Flaccid paralysis bladder & bowel and sustained Priapism Lasts even days till reflex neural arcs below the level recovers.
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Assessment & Managemnt Failure to suspect leads to failure to detect injuries ABCDE – Logroll and remove the spinal board Look for markers of spinal injury Secondary survey Adequate Xray’s Emergency treatment Surgery Definitive care & rehab.
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Clinical features Pain in the neck or back radiating due to nerve root irritation Sensory disturbance distal to neurological level Weakness or flaccid paralysis below the level
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Signs in an Unconcious patients Diaphragmatic breathing (Abdomen expands > Chest) Neurological shock (Low BP & HR) Flexed upper limbs (loss of extensor innervation below C 5 ) Responds to pain above the clavicle only Priapism – may be incomplete.
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Signs of spinal injury Forehead wounds – think of hyperextension injury Localized bruise Deformities of spine - Priapism Beevor’s sign – tensing the abdomen umbilicus moves upwards in D 10 lesions
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Prehospital transfer Awareness of the crew & by A&E staff Modified left lateral position at scene Kendrick or Russell’s extrication device Scoop stretcher slotted together around the patient Agitated patient left alone with hard collar Repeated assessment enroute Head down if they vomit Remove objects from clothes to avoid pressure sores Avoid opiates in high lesions Avoid oral suction in tetraplegics – vagal reflex
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Care in A&E Careful manual handling especially if unconcious Jaw thrust is safer Correct gross spinal deformities Call the anaesthetist if diaphragmatic paralysis or RR>35 Use flexible fibreoptic scopes in unstable fractures Cathetrize to avoid overstretching of detrusor IV fluids – paralytic ileus in first 48hrs. Passive movements to rule out fractures Small iv doses of opiates
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Assessment Document the level of injury Rule out other injuries – DPL in abdominal injuries as there is paralytic ileus and absent peritioneal irritation. Associated injuries in dorsal spine fracture are : - Renal injuries - Chest and Sternal injuries - Wide Mediatinum due to fracture haematoma. - Retroperitoneal injuries
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Radiology Be thorough – Adequacy, Alignment,Bones, Cartilages and soft tissues and distances SCIWORA in kids Low threshold for xray in rheumatoid & Ankylosing spond Flexion injury common in lower cervical spine Extension injury in upper cervical Spine Junction of mobile & fixed part are prone to injury eg. C7 T1 & D12 L1.
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Emergency treatment ABCDE Keep warm Treat if BP<80mmHg & HR <50bpm Spring loaded gardener wells calipers for traction H2 Antagonists & Heparin Methylprednisolone 30mg/kg iv bolus over 15min immediately 45minutes after the bolus a 5.4mg/kg/h infusion over 23 hrs in first 3 hours after the injury. 5.4mg/kg/hr for 47hrs if 4 - 8hrs following the injury.
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