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Published byWalter Augustine Simon Modified over 9 years ago
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Adapted From Temple College EMS Professions
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Poisons Substance which when introduced into body in relatively small amounts causes in structural damage or functional disturbances
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Suspect with: GI signs/symptoms (nausea, vomiting, diarrhea, pain) Altered LOC, seizures, unusual behavior Pupil changes, salivation, sweating, other signs/symptoms of disturbed autonomic nervous system function Respiratory depression Burns, blisters of lips, mucous membranes Unusual breath odors
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Treat Patient, Not Poison Proper support of ABCs is first step in management Contact with Poison Control Center Priority action plan Symptomatic treatments Time management
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Try to determine: What? How much? How long ago? What has already been done? Psychiatric history? Underlying illness?
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When in doubt... Assume containers were full Entire contents were ingested Patient may not be telling you the truth
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If several patients involved... Assume each ingested entire container contents Triage Additional resources
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Always... Bring sample of material if possible Save for analysis, if patient vomits
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Poisoning Management Based on route of entry Ingested Absorbed Inhaled Injected
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Ingested Poisons Prevent absorption of toxin from GI tract into bloodstream Positioning Rapid Transport to definitive treatment center
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Absorbed Poisons Dry chemicals dust skin, then wash Liquid chemicals wash with large amounts of H 2 0 avoid “neutralizing” agents CAUTION Don’t accidentally expose yourself!
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Inhaled Poisons Remove patient from exposure Maximize oxygenation, ventilation CAUTION Don’t accidentally expose yourself!
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Injected Poisons Attempt to slow absorption v Venous constricting bands v Dependent position v Splinting of injected body part v Cold packs (+) [May worsen local injury by concentrating poison]
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Substance Abuse Self administration of a substance in a manner not in accord with approved medical or social practices
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Substance Abuse Psychological dependence Physical dependence Compulsive drug use Tolerance Addiction
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Psychological Dependence Habituation Substance needed to support user’s sense of well-being
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Physical Dependence Substance must be present in body to avoid physical symptoms (withdrawal)
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Compulsive Drug Use Use of drug and rituals/culture associated with its use become an overwhelming desire
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Tolerance Increasing amounts of drug needed to produce same effects Tolerance contributes to addiction by keeping user “chasing the last high”
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Addiction Combination of psychological dependence, physical dependence, compulsive use, and tolerance Patient becomes totally consumed with obtaining, using drug to exclusion of all other things
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Ethyl Alcohol A CNS Depressant Drug
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Ethanol Intoxication Signs Breath odor Swaying, unsteadiness Slurred speech Nausea, vomiting Flushed face Drowsiness Violent, erratic behavior
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Ethanol Clouds signs, symptoms Complicates assessment Head trauma, diabetes, drug toxicity, CNS infection can mimic EtOH intoxication and vice versa
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Patient is NEVER “just drunk” until all other possibilities are excluded
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Experience alcohol withdrawal syndrome if they reduce intake: Restlessness, tremulousness Hallucinations Seizures Delirium tremens--all of above plus tachycardia, nausea, vomiting, hypertension, elevated body temperature Alcohol Addicts
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Life threatening condition! Occurs 1 days to 2 weeks after intake is decreased 5 to 15% mortality Control airway, prevent aspiration, monitor for hypovolemia Delirium Tremens
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Narcotics Opium Opium derivatives Synthetic compounds that produce opium- like effects
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Narcotics Opium Heroin Morphine Demerol Dilaudid v Percodan v Codeine v Darvon v Talwin
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Narcotics Medical Uses analgesics anti-diarrheal agents cough suppressants
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Narcotics Overdose v Coma v Respiratory depression v Constricted (pin-point) pupils
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Narcotics Withdrawal Agitation Anxiety Abdominal pain Dilated pupils –Sweating –Chills –Joint pains –Goose flesh Resembles severe influenza Not a life-threat
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Nembutal Seconal Pentobarbital Amytal Tuinal Phenobarbital Barbiturates
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Induce sleepiness, state similar to EtOH intoxication Medical uses Anesthetics Sedative Hypnotics Barbiturates
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Overdose Coma Respiratory depression Shock Extremely dangerous in combination with EtOH
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Barbiturates Withdrawal Resembles EtOH withdrawal (DTs) Extremely dangerous
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Barbiturate-like Non-barbiturates Doriden, Placidyl, Quaalude, Methyprylon Effects similar to barbiturates Overdose can cause sudden, very prolonged respiratory arrest Withdrawal resembles ETOH; extremely dangerous
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Valium, Librium, Miltown, Equanil, Tranxene Low doses relieve anxiety, produce muscle relaxation High doses produce barbiturate-like effects Tranquilizers
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Overdose: v Unlikely to cause respiratory arrest alone v Extremely dangerous with EtOH Withdrawal Resembles EtOH withdrawal Extremely dangerous Tranquilizers
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CNS Stimulants: Amphetamines Dexedrine, Benzedrine, Methyl amphetamine Relieve fatigue, promote euphoria, reduce appetite
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CNS Stimulants: Amphetamines Overdose Restlessness, paranoia Tachycardia Hypertension CVA, Heart failure Hyperthermia Heat stroke Withdrawal Lethargy Depression
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Stronger stimulant effects than amphetamines Can cause respiratory/cardiovascular failure, heat stroke, lethal arrhythmias CNS Stimulants: Cocaine
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“Snorting” can destroy nasal septum, cause massive nosebleed Airway issue Withdrawal: lethargy depression CNS Stimulants: Cocaine
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LSD, psilocybin, peyote, mescaline, DMT, MDMA Enhance perception Wrong setting may induce “bad trips” with extreme anxiety True toxic overdose rare Hallucinogens
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Phencyclidine PCP, angel dust Produces bizarre, violent behavior Reduces pain sensation Patients may be capable of feats of extreme strength Keep patient in quiet environment, minimize stimulatin
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Glue, paint, gas, light fluid, toluene Inhalation produces state similar to EtOH intoxication Patient may asphyxiate if consciousness lost while “sniffing” Solvents
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Increase risk of arrhythmias May cause liver damage, bone marrow depression Chronic abuse causes CNS damage - paranoia, violent behavior Solvents
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