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اصول EKG
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سیستم هدایتی SA node Pacemaker 60-100 bpm P waves up in I, II, aVF Internodal Pathways AVN; RA contraction Interatrial Pathways LA to depolarize
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سیستم هدایتی AV node Delays impulse by.1s PR segment AV junction AV node & His 40-60 bpm Purkinje/bundles Ventricular depol 20-40 bpm
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سیستم هدایتی
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Repolarization Direction is same as depolarization Autonomic Nervous System (ANS) Sympathetic Nervous System (SNS) Parasympathetic Nervous System (PNS)
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اصول EKG
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اصول EKG: موج P Normal Width <.11 secs Height.5 to 2.5 mm Morphology Flat Biphasic Absent
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اصول EKG: موج P Abnormal Inversions Amplitude P-Pulmonale > 2.5 mm Duration P-Mitrale >.1 sec (or 2 ½ boxes) Absence
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اصول EKG: موج QRS Impulse travels quicker down the left bundle branch (LBB) than the right bundle branch (RBB). Septum depolarizes L to R resulting in a downward deflection Both ventricles are activated simultaneously. Since the RV is smaller, depolarizes quicker resulting in the downward deflection LV depolarizes resulting in the R wave
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اصول EKG: موج QRS
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6 ویژگی موج QRS 1.Duration:.05 -.12 Secs 2.Amplitude: > 5mm; < 20 mm in limb, < 25 in anterior leads 3.Presence of Q waves < 0.04 msec and < 2 mm normal in I, aVL, aVF, V5 4.Axis 5.Progression: Zone of transition V3-V4 6.Configuration
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اصول EKG: موج T و U T waves occur in Same direction as QRS Height: < 5 mm in limb leads, <10 mm in anterior leads U waves After T wave Best seen in lead III Hypothermia/hypokalemia
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اصول EKG
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لیدهای قفسه سینه
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EKG استاندارد EKG استاندارد 12 leads and rhythm strip Limb leads I, II, III, aVR, aVL, aVF Anterior leads V1-V6 Speed = 25 mm/sec Height = 10 mm
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متغیرهای نوار قلب Source Age, Sex Body weight Chest configuration Heart position Food intake Temperature, Exercise Smoking, Hyperventilation Position of precordial leads
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متغیرهای نوار قلب Ideal time for EKG Bayes’ Theorem Normal hearts have abnl EKGs Normal EKG does not r/o heart disease
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رویکرد منظم Rate Rhythm Axis Wave Morphology P, T, and U waves and QRS complex Intervals PR, QRS, QT ST Segment
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تعیین ریت قلب On 6 sec strip, count QRS complexes, X 10 QRS on dark line of tracing, count large boxes, ÷ into 300
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تعیین ریت قلب
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دپلاریزاسیون
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دپلاریزاسیون
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اصول EKG
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موج P در لید II
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دپلاریزاسیون بطن ها در لید قفسه سینه
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محور قلب Find the quadrant Isolate the isoelectric lead Smallest QRS voltage Isolate the perpendicular lead Isolate the vector Double check your findings
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پیدا کردن ربع
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1.1. 3.
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پیدا کردن لید ایزوالکتریک
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پیدا کردن محور
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ریتم / آریتمی Sinus Atrial Junctional Ventricular
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آریتمی سینوسی : معیار و انواع P waves upright in I, II, aVF Constant P-P/R-R interval Rate Narrow QRS complex P:QRS ratio 1:1 P-R interval is normal and constant
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آریتمی سینوسی : معیار و انواع Normal Sinus Rhythm Sinus Bradycardia Sinus Tachycardia Sinus Arrhythmia
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Normal Sinus Rhythm Rate is 60 to 100
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Sinus Bradycardia Can be normal variant Can result from medication Look for underlying cause
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Sinus Tachycardia May be caused by exercise, fever, hyperthyroidism Look for underlying cause, slow the rate
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Sinus Arrhythmia Seen in young patients Secondary to breathing Heart beats faster
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آریتمی دهلیزی : معیار و انواع P waves inverted in I, II and aVF Abnormal shape Notched Flattened Biphasic Narrow QRS complex
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آریتمی دهلیزی : معیار و انواع Premature Atrial Contractions Ectopic Atrial Rhythm Wandering Atrial Pacemaker Multifocal Atrial Tachycardia Atrial Flutter Atrial Fibrillation
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Premature Atrial Contraction QRS complex narrow RR interval shorter than sinus QRS complexes P wave shows different morphology than sinus P wave
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Ectopic Atrial Rhythm Narrow QRS complex P wave inverted
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Wandering Atrial Pacemaker 3 different P wave morphologies possible with ventricular rate < 100 bpm
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Multifocal Atrial Tachycardia 3 different P wave morphologies with ventricular rate> 100 bpm
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Atrial Flutter Regular ventricular rate 150 bpm Varying ratios of F waves to QRS complexes, most common is 4:1 Tracing shows 2:1 conduction
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Atrial Flutter Tracing shows 6:1 conduction
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Atrial Fibrillation Tracing shows irregularly irregular rhythm with no P waves Ventricular rate usually > 100 bpm
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Atrial Fibrillation Tracing shows irregularly irregular rhythm with no P waves Ventricular rate is 40
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Atrial Tachycardia Tracing shows regular ventricular rate with P waves that are different from sinus P waves Ventricular rate is usually 150 to 250 bpm
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P wave May be absent Buried in QRS If present inverted in leads I, II, and aVF Inverted after QRS آریتمی جانکشنال : معیار
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PR interval < 0.12 Secs Rate: Varies Narrow QRS complex آریتمی جانکشنال : معیار و انواع
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آریتمی جانکشنال : انواع Premature Junctional Contractions Junctional Escape Rhythm Accelerated Junctional Tachycardia Junctional Tachycardia Reentrant Tachycardia AVNRT
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Premature Junctional Contractions R-R interval is shorter Beat is early, narrow QRS complex Inverted P wave P wave can be buried in QRS complex
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Junctional Escape Rhythm Junctional origin Rate is 40 to 60
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Accelerated Junctional Tachycardia Junctional origin Rate is 60 to 100
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Junctional Tachycardia Junctional origin Rate is > 100
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Secondary to bypass tract within AV node Premature Atrial Contraction (PAC) depolarizes AV Nodal Reentrant Tachycardia (AVNRT) AV Nodal Reentrant Tachycardia (AVNRT)
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Rate Summary Sinus Tachycardia - 100-160 BPM Atrial Tachycardia - 150-250 BPM Atrial Flutter - 150-250 BPM Junctional Tachycardia - 100-180 BPM
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بلوک های گره AV Delay conduction of impulses from sinus node If AV node does not let impulse through, no QRS complex is seen AV nodal block classes: 1 st, 2 nd, 3 rd degree
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1 st Degree AV Block PR interval constant >.2 sec All impulses conducted
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2 nd Degree AV Block Type 1 AV node conducted each impulse slower and finally no impulse is conducted Longer PR interval, finally no QRS complex
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2 nd Degree AV Block Type 2 Constant PR interval AV node intermittently conducts no impulse
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AV node conducts no impulse Atria and ventricles beat at intrinsic rate (80 and 40 respectively) No association between P waves and QRS complexes 3 rd Degree AV Block
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Caused by bypass tract AV node is bypassed, delay EKG shows short PR interval <.11 sec Upsloping to QRS complex (delta wave) Wolfe-Parkinson-White (WPW)
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Delta wave, short PR interval WPW
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آریتمی های بطنی : معیار و انواع Wide QRS complex Rate : variable No P waves Premature Ventricular Contractions Idioventricular Rhythm Accelerated IVR Ventricular Tachycardia Ventricular Fibrillation
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Occurs earlier than sinus beat Wide, no P wave Premature Ventricular Contraction
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Escape rhythm Rate is 20 to 40 bpm Idioventricular Rhythm
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Rate is 40 to 100 bpm Accelerated Idioventricular Rhythm
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Rate is > than 100 bpm Ventricular Tachycardia
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Torsades de Pointes Occurs secondary to prolonged QT interval
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Unorganized activity of ventricle Ventricular Tachycardia/Fibrillation
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Ventricular Fibrillation
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هایپرتروفی بطن ها و دهلیزها
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Differential Diagnosis Hypertension (HTN) Aortic Stenosis (AS) Aortic Insufficiency (AI) Hypertrophic Cardiomyopathy (HCM) Mitral Regurgitation (MR) Coarctation of the Aorta (COA) Physiologic Left Ventricular Hypertrophy (LVH)
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False positive Thin chest wall Status post mastectomy Race, Sex, Age Left Bundle Branch Block (LBBB) Acute MI Left Anterior Fascicular Block Incorrect standardization Left Ventricular Hypertrophy (LVH)
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Estes Criteria: Diagnosis of LVH
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Right Ventricular Hypertrophy: Causes Chronic Obstructive Pulmonary Disease Pulmonary HTN Primary Pulmonary Embolus Mitral Stenosis Mitral Regurgitation Chronic LV failure
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Right Ventricular Hypertrophy: Causes Tricuspid Regurgitation Atrial Septal Defect Pulmonary Stenosis Tetralogy of Fallot Ventricular Septal Defect
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Right Ventricular Hypertrophy
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Reversal of precordial pattern R waves prominent in V1 and V2 S waves smaller in V1 and V2 S waves become prominent in V5 and V6
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Strain
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Mitral Stenosis Mitral Regurgitation Left ventricular hypertrophy Hypertension Aortic Stenosis Aortic Insufficiency Hypertrophic Cardiomyopathy بزرگی دهلیز چپ : علل
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بزرگی دهلیز چپ : معیار P wave Notch in P wave Any lead Peaks > 0.04 secs V1 Terminal portion of P wave > 1mm deep and > 0.04 sec wide
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Lead II
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موج P: بزرگی دهلیز چپ
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بزرگی دهلیز چپ
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CHD Tricuspid Stenosis Pulmonary Stenosis COPD Pulmonary HTN Pulmonary Embolus Mitral Regurgitation Mitral Stenosis بزرگی دهلیز راست : علل
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Tall, peaked P wave > 2.5 mm in any lead Most prominent P waves in leads I, II and aVF بزرگی دهلیز چپ : معیار
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بزرگی دهلیز راست
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Bundle Branch Blocks بلوک شاخه ای دسته ای
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Normal variant Idiopathic degeneration of the conduction system Cardiomyopathy Ischemic heart disease Aortic Stenosis Hyperkalemia Left Ventricular Hypertrophy بلوک شاخه ای دسته ای چپ : علل
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بلوک شاخه ای دسته ای چپ : معیار Bizarre QRS Morphology High voltage S wave in V1, V2 & V3 Tall R wave in leads I, aVL and V5-6 Often LAD QRS Interval ST depression in leads I, aVL, & V5-V6 T wave inversion in I, aVL, & V5-V6
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Left Bundle Branch Block
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بلوک شاخه ای دسته ای راست : علل Idiopathic degeneration of the conduction system Ischemic heart disease Cardiomyopathy Massive Pulmonary Embolus Ventricular Hypertrophy Normal Variant
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بلوک شاخه ای دسته ای راست : معیار QRS morphology Wide S wave in leads I and V4-V6 RSR’ pattern in leads V1, V2 and V3 QRS duration ST depression in leads V1 and V2 T wave inversion in leads V1 and V2
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بلوک شاخه ای دسته ای راست
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ایسکمی و انفارکتوس
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Normal Complexes and Segments
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J Point
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اندازه گیری
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ایسکمی T wave inversion, ST segment depression Acute injury: ST segment elevation Dead tissue: Q wave
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EKG Changes: Ischemia → Acute Injury→ Infarction
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لیدهای قفسه سینه
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محل لیدهای قدامی V1 Right 4 th ICS V2 Left 4 th ICS V3 Left Sternal border Between V2 and V4 V4 Left MCL 5 th ICS V5 Anterior axillary line 5 th ICS V6 Mid axillary line 5 th ICS
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خونرسانی به قلب Left Anterior Descending (LAD) artery Bulk of LV Anterior wall Apex Part of lateral wall Part of septum
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خونرسانی به قلب Right Coronary Artery (RCA) Right Ventricle Sinus Node 60% of the time Right Atrium Posterior Descending Artery (PAD) 90% of the time
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خونرسانی به قلب Left Circumflex (LCFLX) artery Lateral Wall & Posterior Wall of LV & LA SA node 40% of the time PDA 10% of the time Posterior Descending Artery (PAD) Off RCA 90%/LCFLX 40% AVN, prox BB, IW/PW, basal septum
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View of the Leads II, III, aVF Inferior Wall//RV RCA distribution I, aVL Lateral Wall LCFLX/distal LAD distribution aVR R side of heart V1 & V2 Intraventricular septum Proximal LAD V3 & V4 Anterior wall Mid LAD V5 & V6 Lateral wall Distal LAD
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Standard EKG
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Anterior MI
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Anterior-septal MI
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Inferior AMI
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Right Sided Leads
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Right Ventricular AMI
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Lateral MI
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Posterior Leads
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Posterior AMI
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Inferior-RV-Posterior AMI
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تغییرات موج ST-T
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Strain in Hypertrophy
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Strain in LVH
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Strain in RVH
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Strain vs. Infarction
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پریکاردیت
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تغییرات دیوگسین
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موج T
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خسته نباشید
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