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آخرین ویرایش فرم ارزیابی تخصصی تغذیه
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Nutrition Care Process Steps 1.Nutrition Assessment 2.Nutrition Diagnosis 3.Nutrition Intervention 4.Nutrition Monitoring and Evaluation
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Nutritional care process
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Nutritional Assessment 1.Anthropometric assessment 2.Clinical evaluation 3.Biochemical, laboratory assessment 4.Dietary evaluation
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ESPEN guidelines Questions to be answered: 1.What is the condition now? 2.Is the condition stable? 3.Will the condition get worse? 4.Will the disease process accelerate nutritional deterioration?
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Anthropometric methods in ICU 1.Weight 2.Height estimation 3.Mid-arm circumference 4.Skin fold thickness 5.Head circumference
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Weight
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Ideal Body Weight (kg) 1.Men=48+ 2.3 for each inch over 152 m 2.Women=45.3+2.3 for each inch over 152 cm 3.Correction for skeletal size:
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Add 10% if SS is large Add 10% if SS is large Subtract 10% if SS is small Subtract 10% if SS is small Ideal Body Weight (kg)
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Height in ICU patients Alternative measurements Estimating Height from ulna length
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Estimations of height
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Skin Fold Thickness معرف ميزان چربي زير پوستي و درنتيجه ميزان چاقي خواهد بود. محلهاي اندازه گيري : تريسپس، بايسپس،زير کتف و بالاي تيغه ايلياک. مشکلات عملي : 1. خطاي در اندازه گيري. 2. مشکلات اندازه گيري. 3. وارياسيون توزيع چربي در افراد مختلف ( فردي وجمعيتي ). 4. حساسيت کم.
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Skin Fold Thickness
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Mid arm circumference measured with a nonstretch measuring tape measured with a nonstretch measuring tape midway between the acromion and olecranon of the nondominant arm midway between the acromion and olecranon of the nondominant arm ≤ 15 cm: severe depletion of muscle mass ≤ 15 cm: severe depletion of muscle mass 16–19 cm: moderate depletion 16–19 cm: moderate depletion 20–22 cm: mild depletion 20–22 cm: mild depletion
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Mid arm circumference
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BMI estimation If MUAC is <23.5 cm, BMI is likely to be <20 kg/m2 If MUAC is >32.0 cm, BMI is likely to be >30 kg/m2
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CLINICAL ASSESSMENT
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General: muscle wasting
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Wasting in the hands
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The Shoulder and Elbow The shoulder Normal: rounded or sloped Abnormal: square, can see acromion process The elbow well padded and not showing joint
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The Arm Bend arm and pinch at triceps. Only pinch the fat, not the muscle. Normal: fingers don ’ t meet Abnormal: fingers meet
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The Legs showing muscle wasting
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Essential fatty acid deficiency syndromes (EFADs)
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Zinc deficiency
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Vitamine C Deficiency
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Biochemical Assessment
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Serum Protein levels are not reliable during inflammation
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MUST TOOL Is recommended by ESPEN for community 4 steps. Biochemical markers of nutrition status:
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Plasmatic proteins with short biologic half-life Albumin – -syntetizate in liver, half-life time is 21 days – Normal: 35-45g/l. – Decrease of alb: malnutrition – Trends of changes alb.levels during realimentation are criterium of succesfull terapy. – Acute decrease: acute phase response.
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Biochemical markers of nutrition status: Transferin : syntesized in liver, – biolog HL: 8days. – Value 2-4g/l, RBP: syntesized in liver – Biolog half-life : 12h., – Normal value: 0,03-0,006g/l. – Acute phase reactant (negative)
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Biochemical markers of nutrition status: P realbumin -syntesized in liver, – biolog.half-life:1,5 days. – Normal Value 0,15-0,4g/l. – Decrease in failure of proteosyntesis-indicator of acute protein malnutrition.
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NUTRITIONAL ASSESSMENT Urine urea nitrogen (UUN): to evaluate degree of hypermetabolism (stress level): – 0 – 5 g/d= normometabolism – 5 – 10 g/d = mild hypermetabolism (level 1 stress) – 10 – 15 = moderate (level 2 stress) – >15 = severe (level 3 stress)
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Biochemical parameters Na,K,Cl,Ca,P,Mg, osmolality - blood, urine Acidobasis, lactate urea, creatinin, creatinin clearence, Nitrogen balance bilirubine, ALT, AST, LDH, amylase, lipase cholesterol, triglycerides, glucose – blood, urine
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Other biochemical parameters Trace elements /Zn,Se../ Vitamins Drugs /methotrexate, antiepileptics, antibiotics.../ Aminogram /glutamin../ Interleucins,TNF … Hormones /cortisol, glucagone, adrenaline../.
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Monitoring
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Enteral Nutrition Monitoring: Gastric Residuals Clinically assess the patient for abdominal distension, fullness, bloating, discomfort Place the pt on his/her right side for 15-20 minutes before checking a RV to avoid cascade effect Seek transpyloric access of feeding tube Raise threshold for RV to 200-300 mL Consider stopping RV checks in stable pts
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Some Lab tests
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Na serum levels Hypernatremia: Na over 150 mmol/l hyperaldosteronism hypovolemia renin- angiot-aldost. Hypothalamic damage Hypertonic hyperhydration Diabetes insipidus Brain death
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K serum levels Hypokalemia: K under 3,5 mEq/L – Low intake, bigger uptake, or both – Emesis, diarrhoe / intestinal loss/ – Diuretics – Chemotherapy, antimycotics /renal tubules failure/ – Anabolic phasis – Hyperaldosteronism – Acute metabolic alcalosis
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BUN (5-20 mg/dl ) Consider hydration and Nutrition. High level of urea – high intake of N, – increase catabolism – polytrauma-muscele loss – GIT bleeding – dehydration – low output- renal failure, Low level – malnutrition,serious hepatic failure- ureosyntetic cycle and gluconeogenesis dysfunction, pregnancy- increase ECF
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ALT(SGPT) N V: M: 7-46 F:4-35 U/L High level – – hepatopathologia, – steatosis, – hepatitis, – cell damage,
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AST(SGOT) High level – – hypoperfusion, – hepatitis, – cell necrosis, – muscles damage both aminotransferases increase during damage of hepatic cells during inf.hepatitis.
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TG(10-190 mg/dl ) TG-increase – during sepsis, mainly on the begining, – monitorate during parenter á l nutrition with lipid emulsion Glycemia, serum, urine, Hypoglycemia below 2,5mmol/l-vital danger hyperglycemia- insulin.rezistence, recomendation level of glycemia 4,5-8,2 /2006/ better survive in ICU patient
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Glucose Glycemia, serum, urine, Hypoglycemia below 2,5 mmol/l -(45 mg/dl ) vital danger hyperglycemia- insulin.rezistence Recomendation level of glycemia 4,5-8,2 (80-150 mg/dl )
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P – serum levels(2.7-4.5 mg/dl ) Hypophosphataemia: under 1,9 mg/dl – Acute wastage of energy after succesfully resuscitation, overfeeding sy, anabolism (energetic substrates without K,Mg,P). Hyperphosphataemia – over 5,8 mg/dl – Renal failure – Cell damage
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Urea Urea in urine Increase – catabolism, prerenal failure Decrease – chronic malnutrition, acute renal failure
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Creatinine(0.5-1.1 mg/dl ) Serum levels of creatinine evaluation together with muscle mass, age, gender Increase – bigger offer- destruction of muscle mass, – low output-renal failure Decrease- – low offer-low muscle mass – malnutrition Creatinine clearence, excretion fraction -renal function N-balance – catabolism – the need of nitrogen Uratic acid – cell damage, arthritis uratica
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Monitoring
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Monitoring in PN therapy Variable to be monitoredInitialLater period WeightDailyWeekly serum glucoseDaily3/wk Electrolytes (Na +, K +, Cl - )Daily1-2//wk BUN3/wkWeekly Ca +, P,mg3/wkWeekly Liver function Enzymes3/wkWeekly Serum triglycerides weekly CBC weekly
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Nutritional screening tools
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Monitoring in PN therapy Variable to be monitoredInitialLater period Clinical status Daily Catetheter site Daily Temperature Daily Intake &Output Daily
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