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Chiropractic Manipulative Therapy: Neurological and Orthopedic Basis

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Presentation on theme: "Chiropractic Manipulative Therapy: Neurological and Orthopedic Basis"— Presentation transcript:

1 Chiropractic Manipulative Therapy: Neurological and Orthopedic Basis
Monroe Community Hospital Grand Rounds Presentation Chiropractic Manipulative Therapy: Neurological and Orthopedic Basis Lisa K. Bloom, D.C. Diplomate of the International Board of Chiropractic Neurology Diplomate in Applied Chiropractic Sciences Associate Professor, Diagnosis and Practice New York Chiropractic College Phone:

2 Premise Chiropractic care centers around the maintenance of appropriate movement of the spinal joints and optimizing biomechanics throughout the musculoskeletal system.

3 This refers to discrete areas of hypomobilty within the spinal column
The primary lesion addressed in chiropractic is the vertebral subluxation complex (VSC). This refers to discrete areas of hypomobilty within the spinal column

4 There is a distinct difference between medical and chiropractic use of the term subluxation:
Medical use of the term refers to a hypermobility associated with instability resulting from a loss of integrity of the joint capsules Chiropractic use of the term refers to a hypomobility with no loss of stability

5 VERTEBRAL SUBLUXATION COMPLEX
HYPOMOBILITY ALTERED BIOMECHANICS DECREASED MECHANORECEPTOR ACTIVITY BREAKING OF CELLS - DEGENERATION FIRING OF SPINAL CHEMOSENSITIVE NOCICEPTORS INFLAMMATION DECREASED INHIBITION OF NOCICEPTOR ACTIVITY INCREASED SYMPATHETIC NERVOUS SYSTEM ACTIVITY

6 The purpose of the spinal adjustment is…
to restore normal movement and resolve the resultant biomechanical, physiological and neurological effects of segmental hypomobility.

7 Biomechanical Effects of Hypomobility
Altered intersegmental movement patterns Results in compensatory changes in motor patterns, etc. Creates cellular damage in sites of biomechanical stresses “Immobilization Degeneration…”

8 Loss of normal motion within a joint results in changes in every structural component of the joint; subchondral bone to the synovium, from meninges to the ligamentum flavum. Amiel D, et al. Acta Ortho Scand, 1982 Palmoski M, et al. Arth Rheum, 1979 Paine & Haung. J Neurosurgery, 1972 Lantz C. Chiro Res J, 1988 Enneking & Horowitz. J Bone Joint Surg, 1972 Evans EB, et al. J Bone Joint Surg, 1960

9 Current orthopedic literature recognizes that changes in the pattern of forces transmitted through joints, which occurs during the immobilization process, is universally recognized as contributing to connective tissue degeneration and local changes in the chemical composition of that tissue….

10 We also know that mechanical failure of ligaments, discs, capsules and other connective tissue can result from local variations in chemical composition.

11 Measurable changes within the joint complex...
Chondrocyte loss is measurable within one day Proteoglycan loss is measurable within one week

12 Effects of hypomobility on the intervertebral disc...

13 The nucleus pulposus is the the area of the disc most susceptible to dessication.
The nucleus pulposus is dependent on movement for nutrition and survival...

14 COMPRESSION TRACTION waste out nutrition in disc space waste out

15 Neurological Effects of the Vertebral Subluxation Complex

16 Mechanoreceptors Position sensitive Motion sensitive
Vibration sensitive Pressure sensitive Provide continuous feedback about where the body is in space...

17 MECHANORECEPTOR ACTIVITY
VSC ALTERED BIOMECHANICS COMMUNICATED TO THE CNS VIA MECHANORECEPTORS BRAIN AND SPINAL CORD RESPOND CREATING COMPENSATION IN MUSCLE TISSUE DAMAGE SECONDARY SITES OF ALTERED BIOMECHANICS NOCICEPTOR ACTIVATION INFLAMMATION SYMPATHETIC NERVOUS SYSTEM ACTIVATION

18 Nociceptors Thermo-, mechano-, polymodal and chemosensitive
Spinal nociceptors are almost exclusively chemosensitive Most of the dorsal horn afferents are nociceptors Fire with harmful or potentially harmful stimuli …

19 Pain Not carried by nociceptors Cortical event
Most nociceptive activity never reaches the cortex and the patient will remain symptom-free in the presence of tissue damage

20 Factors that influence the perception of pain
Intensity of the stimulus Duration of the stimulus Descending inhibition

21 SWEAT GLANDS HAIR FOLLICLES DISC
NOCICEPTOR ACTIVITY “PAIN” 10% only! 10% only! CORTEX VSC THALAMUS ALTERED BIOMECHANICS SPINOTHALAMIC TRACT NOCICEPTOR STIMULATION SYNAPSE IN CORD IML ANTERIOR HORN CELL SYMPATHETIC CHAIN MOTOR RESPONSE BLOOD VESSELS SWEAT GLANDS HAIR FOLLICLES DISC ORGAN SYSTEMS

22 Nociceptor Sites Grieve G. Common Vertebral Joint Problems, 1988
Skin Subcutaneous tissue Adipose Joint capsules All spinal ligaments Blood vessels Cancellous bone Periosteum Muscles Tendons Fascia Aponeurosis Dura mater Epidural tissue

23 Nociceptor activity and the sympathetic nervous system…

24 The dorsal horn is a central focal point for mediating autonomic and somatomotor reflexes initiated by nociceptive stimulation. Price D. Psychological and Neural Mechanisms of Pain, 1988; Raven Press, NY.

25 The Effects of Nociceptive Activity
Segmental responses of muscle spasm and sympathetic hyperactivity J. Bonica, & H. Hooshmand, 1993

26 Nociceptive input normally triggers sympathetic activity. Cabell J
Nociceptive input normally triggers sympathetic activity. Cabell J. Sympathetically maintained pain. In: Willis W, ed. Hyperalgesia and Allodynia. Raven Press, NY; 1992.

27 Noxious chemical stimulation of specific spinal structures produce measurable changes in sympathetic nerve activity and visceral function. Budgell B, et al. Spinovisceral reflexes evoked by noxious and Innocuous stimulation of the lumbar spine. J Neuromusculoskel Syst; 1995;3: Cabell J. Sympathetically maintained parin. In: Willis W, ed. Hyperalgesia and Allodynia. Raven Press, NY; 1992.

28 Sensitization of spinal cord neurons primarily by C fibers from muscles, joints and periosteum causes prolonged increased excitability of the nociceptors… C fibers from muscles cause a more prolonged sensation than those innervating skin

29 …over time recruitment of adjacent spinal levels occur which become involved in the complex array of reflexes. Though this facilitation is initiated by somatic afferents, it is sustained by an intrinsic spinal cord process…

30 Results in: Altered somatic function Altered visceral function
Allodynia Sustained pain syndromes Bonica JJ. Clinical importance of hyperalgesia in Hyeralgesia and Allodynia; WD Willis Jr.,ed. 1992; Raven Press, Ltd., NY;

31 Effects of Spinal Nociceptor Stimulation of the Hypothalamus and Reticular Formation
Nausea Pallor Dizziness Faintness Syncope Changes in blood pressure Changes in respiration Changes in heart rate No pain

32 There is good evidence to support the importance of input from spinal afferents in reflex activity of the sympathetic nervous system. Qu, et al, 1988 Meckler and Weaver, 1988 Taylor and Schramm, 1987 Stjernberg, et al, 1986 D. Jordan, 1997 Schramm and Poree, 1991 Hayes, et al, 1991 Weaver and Stein, 1988 Stein, et al, 1989

33 Nociceptor activity can cause reflexive stimulation of the sympathetic nervous system...
This may become chronic without the presence of pain or any other symptom.

34 Spinal Learning (Law of Facilitation)
Altered motor and sensory patterns are “learned” by the nervous system and the musculoskeletal systems

35 Biomechanical stresses  Low grade chronic tissue damage
Release of depolarizing agents (bradykinin, potassium, etc.) Nociceptor stimulation and inflammation Edema Immobilization and myospasm Rebuilding of movement patterns SPONOLEARNING

36 Reasonable conclusions:
Hypomobility is the key ingredient for immobilization degeneration to begin The ID process is progressive if not interrupted Poor biomechanics will damage cells causing dysafferentation Nociceptors will cause the sympathetic nervous system to fire

37 This reflexively facilitates the sympathetic nervous system
Due to nociceptive mediation, pain will come on much later though the degenerative process is measurable within one day/week of the onset of hypomobility

38 Chiropractic Manipulative Therapy: Neurological and Orthopedic Basis
Monroe Community Hospital Grand Rounds Presentation Chiropractic Manipulative Therapy: Neurological and Orthopedic Basis Lisa K. Bloom, D.C. Diplomate of the International Board of Chiropractic Neurology Diplomate in Applied Chiropractic Sciences Associate Professor, Diagnosis and Practice New York Chiropractic College Phone:


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