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LATE COMPLICATIONS OF FRACTURES
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LATE COMPLICATIONS Delayed union Non-union Malunion Joint stiffness
Myoisitis ossificans Avascular necrosis Algodystrophy Osteoarthritis Joint instability Muscle contracture (Volkmann’s contracture) Tendon lesions Nerve compression Growth disturbance Bed sores
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DELAYED UNION Fracture that has not healed in the expected time for type of fracture, patient and method of repair Causes Inadequate blood supply Severe soft tissue damage Periosteal stripping Excessive traction Insufficient splintage Infection
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PERKINS’ TIME TABLE Upper Limb Lower Limb Callus visible 2-3 wks Union
Consolidation 6-8 wks 12-16 wks
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Clinical features X-Ray Persistent pain at fracture site
Instability at fracture site Non weight bearing Disuse muscle atrophy X-Ray Visible fracture line Very little callus formation or periosteal reaction
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Treatment Conservative - To eliminate any possible cause
- Immobilization - Exercise Operative - Indication : Union is delayed > 6 mths No signs of callus formation - Internal fixation & bone grafting
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treatment of delayed union fractures
If alignment is adequate implants are stable but motion exists at fracture sites: apply rigid fixation If alignment is poor: straighten and apply rigid fixation If reduction is inadequate: treat as nonunion
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NON-UNION Fracture has not healed and is not likely to do so without intervention Healing has stopped. Fracture gap is filled by fibrous tissue (pseudoarthrosis)
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causes of nonunion Instability at fracture site
inadequate method of stabilization, inadequate postop care Inadequate blood supply at fracture Poor surgical technique following open reduction, following trauma at time of frature Infection Excessive gap at fracture site Bone loss, distracting force not counteracted by method of fixation, bone loss from ischemia or infection Excessive postop use of limb Use of improper metals or combinations of dissimilar metals Excessive quantities of implants
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Clinical features X-Ray Painless movement at the fracture site
No pain at fracture site Instability at fracture site May be weight bearing with pseudoarthrosis X-Ray Fracture is clearly visible Fracture ends are rounded, smooth and sclerotic Atrophic non-union : - Bone looks inactive (Bone ends are often tapered / rounded) - Relatively avascular Hypertrophic non-union : - Excessive bone formation ` - on the side of the gap - Unable to bridge the gap
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Hypertrophic non-union
Atrophic non-union
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treatment of the 2 types of nonunion fractures.
Vascular nonunion Rigid immobilization Open reduction and compression of fracture with cancellous bone graft Avascular nonunion Surgery required Open medullary canal, debride sclerotic bone Apply rigid fixation Cancellous bone graft
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MALUNION Condition when the fragments join in an unsatisfactory position (unaccepted angulation, rotation or shortening) Causes Failure to reduce a fracture adequately Failure to hold reduction while healing proceeds Gradual collapse of comminuted or osteoporotic bone.
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Clinical features Treatment Deformity & shortening of the limb
Limitation of movements Treatment Angulation in a long bone (> 15 degrees) → Osteotomy & internal fixation Marked rotational deformity Shortening (> 3cm) in 1 of the lower limbs → A raised boot OR Bone operation
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JOINT STIFFNESS Common complication of fracture Treatment following immobilization Common site : knee, elbow, shoulder, small joints of the hand Causes Oedema & fibrosis of the capsule, ligaments, muscle around the joint Adhesion of the soft tissue to each other or to the underlying bone (intra & peri-articular adhesions) Synovial adhesions d/t haemarthrosis
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Treatment Prevention : Joint stiffness has occurred: - Exercise
- If joint has to be splinted → Make sure in correct position Joint stiffness has occurred: - Prolonged physiotherapy - Intra-articular adhesions → Gentle manipulation under anaesthesia followed by continuous passive motion - Adherent or contracted tissues → Released by operation
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MYOSITIS OSSIFICANS Heterotopic ossification in the muscles after an injury Usually occurs in Dislocation of the elbow A blow to the brachialis / deltoid / quadriceps Causes (thought to be due to) muscle damage Without a local injury (unconscious / paraplegic patient)
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Clinical features X-Ray Pain, soft tissue tenderness Local swelling
Joint stiffness Limitation of movements Extreme cases: - Bone bridges the joint - Complete loss of movement (extra-articular ankylosis) X-Ray Normal Fluffy calcification in the soft tissue
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Treatment Early stage : Joint should be rested
Then : Gentle active movements When the condition has stabilized : Excision of the bony mass Anti-inflammatory drugs may ↓ joint stiffness
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AVASCULAR NECROSIS Circumscribed bone necrosis Common site : Causes
Interruption of the arterial blood flow Slowing of the venous outflow leading to inadequate perfusion Common site : Femoral head Femoral condyls Humeral head Capitulum of humerus Scaphoid (proximal part) Talus (body) Lunate
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Conditions associated with AVN
Perthes’ disease Epiphyseal infection Sickle cell disease Caisson disease Gaucher’s disease Alcohol abuse High-dosage corticosteroid
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Clinical features X-Ray Joint pain, stiffness, swelling
Restricted movement X-Ray ↑ bone density Subarticular fracturing Bone deformity
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Treatment Avoid weight bearing on the necrotic bone
Revascularisation (using vascularised bone grafts) Excision of the avascular segment Replacement by prostheses
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ALGODYSTROPHY (COMPLEX REGIONAL PAIN SYNDROME)
Previosly known as Sudeck’s atrophy Post-traumatic reflex sympathetic dystrophy Usually seen in the foot / hand (after relatively trivial injury) Clinical features Continuous, burning pain Early stage : Local swelling, redness, warmth Later : Atrophy of the skin, muscles Movement are grossly restricted
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X-Ray Patchy rarefaction of the bones (patchy osteoporosis)
Algodystrophy
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Treatment Physiotherapy (elevation & active exercises) Drugs
- Anti-inflammatory drugs - Sympathetic block or sympatholytic drugs (Guanethidine)
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OSTEOARTHRITIS Post-traumatic OA secondary OA
Joint fracture with severely damaged articular cartilage Within period of months secondary OA Cartilage heals Irregular joint surface may caused localized stress → secondary OA Years after joint injury
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Clinical features Treatment Pain Stiffness Swelling Deformity
Restricted movement Treatment Pain relief : Analgesics Anti-inflam agent Joint mobility : Physiotherapy Load reduction : wt reduction Realignment osteotomy (young pt) Arthroplasty (pt > 60yr)
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Thank You
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