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Published byGertrude Rich Modified over 9 years ago
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Complication o Labor
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Psychologic Disorders Alterations in thinking, mood or behavior Keep her well oriented and promote optimal functioning in labor. Focus on maintaining safe environment and ensuring fetal and maternal well-being
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Dystocia r/t dysfunctional contractions Accounts for ~ 50% C/S for primips; <5% C/S for multips Hypertonic: in 1 st phase- poor quality U/Cs, become more frequent, but ineffective and changing dilatation or effacement prolonged latent phase Tx: sedation, oxytocin, amniotomy Hypotonic: irreg, low amplitude protracted labor and arrest of dilatation Tx: oxytocin, amniotomy
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Active Management of Labor Standardized criteria for diagnosis of labor Standardized method of labor management One-to-one nursing care in labor Prenatal education to teach re: this protocol Method: Amniotomy right away VE frequently If change not as expected, oxytocin
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Precipitous Labor and Birth From beginning of regular contractions to delivery is 3 hours or less Risks: Abruption Cervical and perineal lacerations Fetal head trauma Women with history may be scheduled for induction
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Post-term Pregnancy > 42 completed weeks Cause of true post-term is unknown; often incorrect dates Maternal Risks: Large baby and associations Psychologic ills Fetal-Neonatal Risks: Placental changes insufficiencies Oligohydramnios macrosomia birth trauma, glucose maintenance problems Meconmium stained fluid (aspiration) As pregnancy approached term, fetal well-being studies done
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Fetal Malposition OP position: Fetus must rotate 135° or occasionally born in OP position If born OP, increased risk of 3 rd or 4 th degree laceration, broken symphysis May use forceps or manual rotation Positioning: knee chest, pelvic rocking
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Fetal Malpresentation Brow Usually C/S recommended Perinatal morbidity and mortality: Trauma: cerebral and neck compression; damage to trachea and larynx Tx: pelvimetry, oxytocin?, C/S Face Perinatal morbidity and mortality: Risk of prolonged labor, fetal edema, swelling of neck and internal structures, petechiae, ecchymosis Tx: C/S in no progress
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Fetal Malpresentation Breech Most common malpresentation Frank breech most common Risk of cord prolapse; fetal anomolies 3x higher If vag del: head trauma, fetal entrapment Tx: external version (50-60% success), if vag del: epidural, double set-up
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Fetal Malpresentation Shoulder Version may be attempted C/S Compound presentation
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Macrosomia >4500 g Obese 3-4x more likely to have macrosomic baby ↑ risk of perineal lacerations, infection Most significant problem is shoulder dystocia OB emergency permanent injury of brachial plexus, fx clavicle, asphyxia, neurologic damage Tx: Assessment of adequacy of pelvis Suprapubic pressure Intentional breaking of clavicle ?C/S
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Multiple Gestation Mother at risk for: Hypertension or preeclampsia Anemia Hydramnios PPROM, IUGR, incompetent cx Malpresentation More physical discomforts
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Multiple Gestation Tx: U/S to diagnose amnion/chorion, follow growth, observe for twin-twin transfusion Frequent office visits to monitor for problems Likely to deliver by C/S
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Abruptio Placentae Premature separation of normally implanted placenta from the uterine wall Very high mortality Cause unknown but r/t Maternal hypertension Maternal trauma Cigarettes, cocaine Short umbilical cord, high parity More common in Caucasian and African American than Asian or Latin American
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Abruptio Placentae
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http://video.about.com/pregnancy/Pla centa-Abruptio.htm
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Abruptio Placentae Classification O=asymptomatic, diagnosed after birth I=mild, most common II=mod, both mom and baby show signs of distress III=severe, maternal shock and fetal death likely
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Abruptio Placentae Types Marginal-blood passes between fetal membranes and uterine wall and escapes vaginally; separation at periphery of placenta Central-separates centrally, blood trapped between placenta and uterine wall. No overt bleeding Complete-massive vaginal bleeding in presence of almost total separation
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Abruptio Placentae
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Blood invades myometrial tissue pain and uterine irritability. May necessitate hysterectomy after delivery secondary to inability to uterus to contract. May lead to coagulation defects
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Abruptio Placentae Maternal Risks Blood coagulation problems Shock Renal failure (r/t hemorrhage) Possible hysterectomy Fetal-Neonatal Risks If separation ~50% 100% demise Depending upon separation, time before delivery, maturity of baby neurologic damage
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Abruptio Placentae Tx Continuous EFM (if baby alive) Develop plan for birth Maintain CV status/tx hypovolemic shock Follow blood coag studies/have blood factors available
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Placenta Previa Improperly implanted in lower uterine segment Types Low lying: close proximity to os, but doesn’t reach it Marginal: edge of placenta at margin of the os Partial: internal os is partially covered by placenta Total: internal os completely covered
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Placenta Previa
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Cause unknown, but associated with Multiparity Increased age Defective development of blood vessels in decidua Defective implantation of the placenta Prior C/S Smoking Large placenta
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Placenta Previa Tx Continuous EFM Differential diagnosis ☺No vag exam until previa r/o (U/S, other assessments) Care depends on amt bleeding, gestational age, assessment of fetus
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