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Electrical Injury
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Electrical Injury In the U.S. 52,000 admissions/yr
3-8 % of all burn unit admissions May-Sept lightning related. Decrease in incidence due to GFCIs
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Electrical Injury - Epidemiology
Ages yrs. High voltage mostly occupational injury 20% Children Low voltage injuries in toddlers M:F 1.7:1 High voltage injuries in adolescents % male
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Electrical Injury - Pathophysiology
Electrical – tetany, arrhythmia Thermal – burns, coagulation Mechanical – fractures, dislocation
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Ohm’s Law I= V/R I= current V= voltage R= resistence
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Joule’s Law E=I²RT E= energy I= current R= Resistence T= time
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Electrical Injury - Pathophysiology
Current pathway defines resistence - Vertical higher incidence of complication - Hand – to – hand pathway - Below symphysis, stradle pathway
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Electrical Injury - Classification
High (>1000 Volt) vs. low (<1000 Volt) voltage Direct (lightning) vs. alternating (50 Hz) current Arc injury (high temperature), flashover
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Cardiovascular Involvment
Mostly in vertical injury DC – Asystole AC High VF/ VT, asystole Low ectopic beats, AF, tachycardia, bradycardia, ECG changes Coagulation necrosis, coronary spasm, MI
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Respiratory Involvement
Tetany of respiratory muscle Brain stem injury May induce hypoxia, acidosis cardiac arrest
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Nervous System Immediate - loss of consciousness, amnesia
Early - intracranial hemorrhage, vertebral fractures Late - ALS, transverse myelitis, ascending paralysis Peripheral neuropathy, RSD
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Vascular Injury Large arteries – medial necrosis, aneurisms
Small vessels – intimal injury, coagulation necrosis Secondary to compartment syndrome
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Limb Injury Dislocations and fractures Coagulation of blood vessels
Muscle ischemia and edema Compartment syndrome Thermal injury from bone heating Infection clostridial, streptococcal
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Other Injuries GI – ileus, stress ulcers, direct injury
Ophthalmic – cataract, iridiocyclitis, autonomic injury Otologic – tympanic membrane perforation, vertigo, sensoryneural injury
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Injury Characteristics
Low Voltage 77% YO 60% extremity 40% oral commisure No mortality Complete functional recovery High Voltage 76% YO 33% limb amputations 30% deep muscles 12% fasciotomy/ escharotomy No mortality
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Electrical Injury - Management
Combined ATLS + ACLS protocols Cardiac monitoring for 24 hrs if LOC, ECG changes or arrhythmias IM dT IV H2 - blockers
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Electrical Injury – Resuscitation
1.7 X Parkland formula or 9 ml/kg/%TBSA Urine output ml/hour Clearance of any pigment in urine Bicarbonate - blood pH > 7.45 Osmotic diuresis – IV MANNITOL 25 gr
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Electrical Injury – Wound Managemant
“True” high tension Sharply demarcated Always full thickness Leathery appearence
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Electrical injury – Wound Management
“Progressive necrosis” theory Primary resuscitation. Early exploration and debridment “Second look” in hrs –definitive Tx Primary closure Coverage Amputation
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Wound Management – Extremities
Frequent envolvement of the hand Exit point in one or both legs Arc injury in distal fore arm or axilla
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Wound Management – Extremities
Initial assessment usually predicts outcome: Depth of burns Ischemia Anasthesia Flexion position Muscle viability- response to electrocautery
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Electrical Burn - Extremities
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Wound Management – Extremities
Exploration - large volume underlying necrotic area Full thickness burns Proximal periosseous myonecrosis Retained questionable tissue may lead to contamination and further compromise
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Wound Management - Scalp
Saucer shaped, deapest in the middle Delayed Tx osteomyelitis and epidural abscess Debridment of soft tissue, outer cortical bone and skin grafting Full thicknss skull - devitalization & flap coverage
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Wound Management – Trunk & perineum
Suspect visceral injury Lung – Atelectasis and edema Abdomen – consider as penetrating wound Perineum –urinary and bowel diversion & debridment +STSG
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Electrical Injury -Summery
סוג הפגיעה ומיקומה טיפול ראשוני לפי פרוטוקולים ACLS ו- ATLS החייאת נוזלים אקספלורציה והטרייה מוקדמים טיפול דפיניטיבי מוקדם – בכל שיטות השחזור המקובלות
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