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Published byLisa Prudence Merritt Modified over 9 years ago
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Intussusception Rory Murphy
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History. HPC 80 ♂ 4/7; general malaise. 1/7; nausea, profuse vomiting, diarrhoea & “crampy” lower abdominal pain. Recent campylobacter gastroenteritis. PMHx Chronic Renal Failure. (Dialysis* 3 a week.). HTN
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Physical Exam. Vitals. HR 80 Hypertensive Apyrexial SaO2 95% Abdominal, Soft Non-tender. No gaurding, rigidity or organomegaly, Hernial orifices clear. BS+ PR/FOB not documented
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Initial Investigations. Bloods. Hb 13 WCC 7.78 Neutophils 6.7 Urea 31 Na 134 Creat 848 LFT’s NAD Amylase CRP 26 Stool Culture,Ova&Oocytes. Neg Imaging. PFA: No gaseous distension of the bowel. Abdominal U/S: NAD. OGD; Hiatus Hernia Mild Antral Gastritis
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Initial Differential Diagnosis and Treatment. Post-Infectious gastroenteritis/Malabsorptive State. Nausea and Vomiting intermittently. 9 days post admission symptoms improved. Resolved gastroenteritis. Aspiration pneumonia.
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Repeat Investigations. PFA x 2 revealed dilated loops of small bowel. However clinical discordence patient passing bowel motions and non distended.
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Intra-operative Images.
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Intussusception exists when a proximal segment of bowel (intussusceptum) telescopes into the lumen of the adjacent distal segment. 1 % of all bowel obstructions, 5% of all intussusceptions, Adult Intussusception.
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Intussusception is a different entity in adults than it is in chlidren. Pathology is found in 70% to 90% of cases in the adult population. Intraluminal lesions alter normal bowel peristalsis and form leading edges for the intussusceptum. 9'0
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Adults may present with acute, intermittent, or chronic reported problems." The predominant symptoms usually are those of bowel obstruction and, consequently,intussusception often is misdiagnosed initially in the adult population.
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