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Published byCory Burns Modified over 8 years ago
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DYSMENORRHEA AHMED ABDULWAHAB CONSULTANT ASSISTANT PROFESSOR
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Classification. Primary dysmenorrhea. Occurs during ovulatory cycles, and usually 6 to 12 month after menarche. Etiology. Uterine contractions with ischemia and increase contractility, and resting tone due to prostaglandin production. There is associated symptoms like nausea vomiting and headache. Unovulatory cycle contain little PGs and hence painless.
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Clinical features Cramping pain usually begin few hours before the cycle and may persist for hours or days. It is localized in the lower abdomen and may radiate to the thigh and lower back May be associated with altered bowel habits, nausea,fatigue, dizziness, and headache.
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treatment NSAIDs are highly effective, ibuprofen and mefenamic acid may cause GIT disturbance. Other class the cyclo-oxygenase inhibitor that have little effect on GIT. Oral contraceptive pills by inhibiting ovulation. Progestins. Tocolytic.
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Secondary dysmenorrhea. It is secondary to an underlying cause and PGs may play a role. Clinical features. Generally the pain is not limited to the menses and can occur before as well as after the menses. It develops in older women and usually associated with other symptoms like dyspareunia, infertility and abnormal uterine bleeding.
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Causes. 1-Endometriosis. Pain in pre and postmenstrual phase,there is deep dyspareunia and tender pelvic examination. Pelvic inflammation, like endometriosis initially pain may be menstrual and often extend into the premenstrual phase with each cycle, may have inter menstrual bleeding.
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2-Fibroid and adenomyosis. There is dull dragging sensation, uterus is clinically enlarged and may be tender. 3-Ovarian cysts. Should be clinically evident. 4-Pelvic congestion. There is dull ill-defined pelvic ache, usually worse pre-menstrually and relived by menses.
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Treatment. Treatment of the underlying disease. The treatment used for the primary dysmenorrhea are often helpful.
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