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How can we preserve the visual function in idiopathic intracranial hypertension?
Hlavacova P., Vlkova E., Doskova H. FN Brno, Department of Ophthalmology
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Case report 1 34-year old obese woman
Headaches, pulsatile tinnitus, visual obscurations of the right eye Neurological status and MRI scan: no patology Opening pressure of cerebrospinal fluid (CSF) 400 mm H2O; CSF: no pathology Visual acuity (VA): OD 0,9; OS 1,0 Perimetry: OD blind spot enlargment, OS physiological
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18 months after the surgery VA is 1,0 and VF is physiological
Therapy: Acetazolamide 1,5g/day Optic nerve sheaths decompression (ONSD) of the right eye 18 months after the surgery VA is 1,0 and VF is physiological
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Case report 2 51-year old healthy man
Headaches, pulsatile tinnitus, visual obscurations of the right eye Neurological status and MRI scan: no pathology, Opening pressure 700 mm H2O; CSF: no pathology VA OD 0,9; OS 1,25 Perimetry OD blind spot enlargement, OS physiological
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Therapy: ONSD Acetazolamide 3g/day failed and ventriculoperitoneal shunt input 18 months after the surgeries VA is 1,0 and perimetry showed enlargement of blind spot
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Case report 3 54-year old woman
Idiopathic intracranial hypertension for 4 years, therapy: Acetazolamide 250 mg/day Progressive visual loss of both eyes, esp. left eye Intermittent headaches, pulsatile tinnitus Opening pressure 500 mm H2O; CSF: no pathology Neurological status: no pathology, MRI scan: the slit ventricles VA OD 0,9, OS light perception Perimetry OD tapered nasally to 30˚, OS concetric tapering of VF to 20˚
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Valve and programmer of LP shunt
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12 months after the surgeries VA and perimetry remain unchanged.
Therapy : ONSD of the right eye Acetazolamide 3 g/day failed and LP shunt input 12 months after the surgeries VA and perimetry remain unchanged.
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Conclusion: ONSD appears to be an efficient method in early treatment of papilloedema in IIH There are different ways how can we preserve visual function in IIH. Patient´s compliance and neurosurgical cooperation is very important.
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