1. General features of insulin signal transduction pathways Jeong-a Kim, et al. Circulation 2006;113:1888-1904

2. Insulin-stimulated secretion of ET-1 from vascular endothelial cells is mediated by MAP-kinase (MAPK)- dependent pathways but not PI 3-kinase–dependent pathways Jeong-a Kim, et al. Circulation 2006;113:1888-1904

3. Parallel impairment in insulin-signaling pathways under pathological conditions contributes to synergistic coupling of insulin resistance and endothelial dysfunction Jeong-a Kim, et al. Circulation 2006;113:1888-1904

4. Shared and interacting mechanisms of glucotoxicity, lipotoxicity, and inflammation Jeong-a Kim, et al. Circulation 2006;113:1888-1904

5. Pathway-specific insulin resistance creates imbalance between prohypertensive and antihypertensive vascular actions Jeong-a Kim, et al. Circulation 2006;113:1888-1904

6. Vasodilator actions of insulin and DHEA mediated by NO are regulated by PI 3-kinase (PI3K)-dependent signaling pathways Jeong-a Kim, et al. Circulation 2006;113:1888-1904

7. In vitro model of metabolic insulin resistance with compensatory hyperinsulinemia in vascular endothelium Jeong-a Kim, et al. Circulation 2006;113:1888-1904

8. Mechanisms for the contribution of insulin resistance to atherosclerosis Jeong-a Kim, et al. Circulation 2006;113:1888-1904

9. SHRs serve as a model of the metabolic syndrome Jeong-a Kim, et al. Circulation 2006;113:1888-1904

10. Losartan therapy simultaneously improves adiponectin levels (A), insulin resistance (B), endothelial dysfunction (C), and inflammatory markers (D) in subjects with hypercholesterolemia and hypertension Jeong-a Kim, et al. Circulation 2006;113:1888-1904 QUICKI indicates Quantitative Insulin-Sensitivity Check Index