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Heart Anatomy + ECG Aaqid Akram MBChB (2013) Clinical Education Fellow
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Brachiocephalic Trunk L Subclavian Artery L Common Carotid Artery Arch of the Aorta Superior Vena Cava Pulmonary Artery L Pulmonary Vein L Atrium Mitral Valve (Bicuspid) Chordae Tendinae Papillary Muscle Endocardium Myocardium Septum Aortic Valve (Semilunar) Inferior Vena Cava L Ventricle R Ventricle Tricuspid Valve Pulmonary Valve R Atrium Fossa Ovalis
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Ligamentum Arteriosum R Coronary Artery Circumflex Artery L Anterior Descending Artery
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Cardiac Cycle
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Introduction Wash Hands Introduce yourself Confirm patient + ALLERGY STATUS Explain investigation to patient Gain verbal consent Offer chaperone (Chest will be exposed) – If opposite sex you require a chaperone for your own safety
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The ECG Machine Power (plugged/battery) Demographics Paper All leads intact Stickers available Scale – vertical axis (0.1mV = 1mm = 1 small square)
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Placing Stickers
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There’s only 10 leads…. How can it be a 12 lead ECG?
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Interpreting an ECG Demographics Obvious abnormality Rate Rhythm Axis P wave PR Interval QRS Complex ST segment T wave Summary
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Rate 1500 small squares (0.04 seconds) = 60s No of small squares between R-R = x 1500/x = ventricular rate per minute If normal calibration rhythm strip = 50 large squares (0.2seconds) = 10 seconds Count QRS complexes on rhythm strip Multiply by 6 = ventricular rate per minute
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Rhythm Sinus = p wave before every QRS Complex Regular = QRS complexes equidistant – Mark 3 R-R points on the edge of a paper – Move to next three complexes – Do the marks on the paper correlate to the R waves?
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Axis
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P Wave T Wave
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P wave Atrial depolarisation (Sino Atrial Node) 2-3 mm high 0.06 – 0.12 seconds duration Usually positive deflection throughout ECG Peaked/enlarged = atrial hypertrophy Inverted = retrograde/reverse conduction Absent = conduction by route other than SA
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PR Interval Impulse from atria to AV Node, Bundle of His, bundle branches 0.12 – 0.2 seconds duration Short = impulse did not originate from SA Long = AV Block
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1 st Degree Heart Block 1 st Degree: – QRS complex after every P wave – Prolonged PR Interval – No Rx necessary unless symptomatic
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2 nd Degree Heart Block Mobitz Type 1 (Wenckebach): – Each successive impulse from SA node delayed slightly longer than previous impulse – A QRS complex is dropped – Cycle repeats
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2:1 Heart block xx x x xx Mobitz Type 2: – Occasional SA impulses fail to cause ventricular depolarisation – Regular P waves, but some dropped QRS complexes 2 nd Degree Heart Block
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3 rd Degree Heart Block Complete Heart Block: – Impulses from atria cannot pass the AV node – Atria depolarise independently to ventricles – Life threatening
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QRS Complex Deep wide Q waves may suggest old infarct Total duration <0.12 seconds >0.12 seconds = ventricular conduction delay
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Bundle Branch Block Bundle branch fails to conduct impulses Ventricles contract at slightly different times Block further down the bundle = hemiblock Cell-cell conduction slower than via specialised pathway therefore prolonged depolarisation New Left Bundle Branch Block = ACS
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QT Interval Time from ventricular depolarisation to ventricular repolarisation Varies according to heart rate QTc = corrected QT interval to 60bpm Males <450 ms / Females <470 ms Prolonged QT interval increases risk of life threatening arrhythmias
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Torsades de Pointes
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Drugs affecting QT Interval DrugType AmiodaroneAntiarrhytmic AmitriptyllineAntidepressant ChlorpromazineAntipsychotic/antiemetic ClarithromycinAntibiotic DroperidolSedative/antiemetic ErythromycinAntibiotic FluoxetineAntidepressant HaloperidolAntipsychotic KetoconazoleAntifungal LevofloxacinAntibiotic MethadoneOpiate agonist QuinidineAntiarrhythmic SertralineAntidepressant SotalolAntiarrhythmic SumatriptanAnti migraine
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ST Segment Segment affected if acute ischaemia/infarction Elevation = >1mm Depression = >0.5mm
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T Wave Ventricular repolarisation Usually upright deflection Tented T waves = hyperkalaemia/myocardial injury Inverted T wave = ischaemia Camel Hump = hidden P/U wave
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Summary Present all positive findings and important negative findings. Advise on urgency of management.
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Supraventricular Tachycardia
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Atrial Flutter
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Atrial Fibrillation
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Ventricular Tachycardia
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Ventricular Fibrillation
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Asystole
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Any Questions? Thank You
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