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1 Aging and Exercise Brooks Chpt 32 Brooks Chpt 19 Rogers and Evans
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2 Aging and Exercise It is dificult to quantify the effects of aging on physiological function and physical performance. Some people physically deteriorate with age due to a lack of exercise, obesity, poor diet, smoking, and stress. Some individuals are active and are still fit in their 50s, 60s and 70s.
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3 Aging and Exercise Disease further complicates our understanding of the aging process. osteoarthritis, atherosclerosis Physiological systems vary in the extent to which they deteriorate with age. It is difficult to separate deconditioning and disease from the aging process.
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4 Aging and Exercise Lifestyle (diet, exercise) will influence performance and health during aging, but they will not halt the aging process. Life expectancy in 1900: 47 years 2000: 76 years The maximum lifespan (100) has not changed Quality of life is very important. North Americans only have healthy quality life during 85% of their lifespan.
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5 Response to Training Older people readily adapt and respond to endurance and strength training. Endurance training helps maintain cardiovascular function, enhances exercise capacity, and reduces factors associated with heart disease, diabetes, some cancers. Strength training prevents loss of muscle mass and strength with aging.
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6 Response to Training Exercise prevents bone loss and improves postural stability, which the risk of fractures and falling. Mobility exercises improve flexibility and joint health. Exercise also provides psychological benefits. Exercise does not retard the aging process, it just allows the person to perform at a higher level.
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7 The Aging Process Physiological control mechanisms are impaired in older people. reaction time resistance to disease work capacity recovery time body structures are less capable and resilient Very fit older individuals are still prey to the ravages of old age.
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8 The Aging Process Genetic factors have a profound influence on the length of life, while environmental and genetic factors govern quality of life. Life spans of identical twins are remarkably similar (2 - 4 yrs of each other). nonidentical twins (7 - 9 yrs)
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9 The Aging Process Peak physiological function occurs at about 30 years of age. After 30, most physiological factors decline at a rate of 0.75 to 1.0% a year. VO 2 max, CO, strength, power, flexibility neural function, body fat table 32-2; physiological effects of aging All of these factors can be positively affected by exercise, especially if training has been maintained throughout life.
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10 Maximal Oxygen Consumption VO 2 max approx 30% between 20 and 65 rate of decline is greatest after age 40 decreases in VO 2 max is quite variable VO 2 max due to ’s in HR max, SV, power output, fat-free mass, a-vO 2 difference.
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11 Heart Rate During submax exercise, HR is lower with age at any relative exercise intensity, but the same at any absolute intensity. Cardiovascular drift is greater with age. Longer recovery rate following exercise. -adrenergic responsiveness with age, this will HR max. Age related in HR max is partially due to deconditioning and loss of muscle mass.
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12 Stroke Volume and Cardiac Output Aging impairs the heart’s capacity to pump blood. CO and SV is in older adults during ex. Gradual loss of contractile strength due to in Ca 2+ -myosin ATPase activity and possible myocardial ischemia. The heart wall stiffens, which delays ventricular filling ( SV). Rely more on Frank-Starling mechanism.
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13 Stroke Volume and Cardiac Output The elasticity of the major blood vessels and the heart due to connective tissue changes. Heart mass usually and there are fibrotic changes in the heart valves with age. Vascular stiffness the peripheral resistance to blood flow, which afterload of the heart. peripheral resistance also raises SBP at rest and during exercise (no change in DBP).
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14 Arteriovenous Oxygen Difference a-vO 2 with age, which aerobic capacity. from 16 vol%(20yrs) to 12 vol%(65yrs). The in a-vO 2 is due to: fiber/capillary ratio total hemoglobin respiratory capacity of the muscle in muscle mito mass oxidative enzymes
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15 Circulatory Regulation The capacity of the autonomic reflexes that control blood flow are diminished with age. in circulation to the skin in orthostatic tolerance in the elderly
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16 Endurance Training Endurance training produces improvements in aerobic capacity in older individuals that are similar to those seen in younger individuals. 6 months of endurance training can improve VO 2 max by 20%. Endurance training: submaximal HR resting and exercise SBP faster recovery HR SV, CO
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17 Endurance Training The rate and magnitude of CV adaptation to endurance exercise training depends on the type, intensity, frequency, and duration of exercise, as well as genetic factors. Older women do not have the same in SV as men with training. May be due to lack of estrogen in postmenopausal women. They have similar improvements in VO 2 max as men, due to a larger a-vO 2 difference.
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18 Endurance Training Assessing the effects of age on athletic performance is difficult. As more masters athletes compete, the limitations produced by age will become more apparent. Elderly individuals require a VO 2 max of 20 ml·kg -1 ·min -1 for an independent lifestyle. A well structured endurance training program can the fitness of an older person to this level.
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19 Pulmonary Function The lung has a large reserve capacity to meet ventilation requirements during exercise. This reserve capacity gradually between 30 and 60 years (faster in smokers). Pulmonary function changes: size of the alveoli ( vascularization) elasticity of support structure work of breathing weakening of respiratory muscle
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20 Pulmonary Function The deterioration in pulmonary function is similar in magnitude to that in the CV system. Unless the in pulmonary function is by disease, ventilation remains adequate during exercise in older individuals. Ventilation does not limit endurance performance. Training maximum ventilation.
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21 Skeletal System Bone loss is a serious problem in older people, particularly women. Women begin to lose bone mineral at 30 and men at 50 years of age. Estrogen deficiency in postmenopausal women is though to accelerate bone loss in women over 45. Although the exact mechanism of bone loss is not completely understood, the contributing factors are: inactivity, diet, skeletal blood flow and endocrine function.
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22 Skeletal System Exercise is important in the prevention and treatment of osteoporosis. Bones become stronger when stresses are placed on them. Elderly male athletes have higher bone mineral content and density than non-athletes. Excessive training (over training) can bone mineral density. Lifelong participation in physical activity may provide a buffer against bone loss.
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23 Joints Joints become less stable and less mobile with age. Aging is associated with: degradation of collagen fibers; cross-link formation; fibrous synovial membranes; joint surface deterioration; viscosity of synovial fluid. It is difficult to separate aging from accumulated wear and tear. Trauma to the joint cartilage results in formation of scar tissue (impairs ROM). ROM exercises can flexibility.
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24 Skeletal Muscle Loss of muscle mass and strength can have severe effects on the quality of life of the elderly. Atrophied muscle contribute to obesity by metabolic rate. Poor muscular support chance of falls.
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25 Strength Muscle strength decreases appox 8% a decade after the age of 45. Aging results in a in isometric and dynamic strength and speed of movement. Strength losses are due to: size and # of muscle fibers atrophy or loss of type II fibers in the respiratory capacity of muscle in connective tissue and fat
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26 Muscle Fiber Types With age there is a selective loss of type II fibers, which available strength and power. Type II fibers more rapidly in the lower than upper body. The mechanisms involved in muscle contraction are also impaired: less excitable, greater refractory period conc of ATP and CP are maximum contractile velocity
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27 Muscle Biochemistry There is loss of biochemical capacity with age. in glycolytic enzymes (LDH). There are no changes or slight in oxidative enzymes. Relative strength with training are similar in young and old individuals.
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28 Exercise and Cardiovascular Disease Risk Factors Exercise (even low intensity) has very significant health benefits in the elderly. Exercise blood insulin levels and improves glucose tolerance and insulin sensitivity. Exercise resting and exercise SBP. Exercise improves plasma lipid profiles: plasma triglyceride and cholesterol; HDL.
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29 Body Composition and Stature Body composition and stature change markedly with age. Body weight from age 20 to 60 and then . Weight gain is due to an in % body fat. A greater proportion of body fat is stored internally rather than subcutaneously. Stature with age: rounding of the back; disc compression; vertebrae deterioration. Exercise is very important in managing body composition in the elderly.
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30 Neural Function Many neurophysiological changes occur with aging. Age associated motor unit remodeling. Some denervated type II fibers become reinnervated by adjacent type I fibers. Neural changes include: visual acuity; hearing loss; short-term memory; information processing; reaction time. Physical training has little effect on the deterioration of neural function.
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31 Exercise Prescription for the Elderly The principles of exercise prescription are the same for everyone, however caution must be taken with the elderly to the risk of injury. Elderly individuals have more ECG abnormalities during exercise. Care must be taken when determining the type and intensity of exercise. Maximum heart rates vary considerably in the elderly (HR max : 105 - 200 for 60yr olds).
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32 Exercise Prescription for the Elderly Basic principles for exercise prescription with the elderly: Careful progression in intensity and duration. Warm up slowly and carefully. Cool down slowly. Static stretching after exercise.
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