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EGFR Mutation-Induced Alternative Splicing of Max Contributes to Growth of Glycolytic Tumors in Brain Cancer Ivan Babic, Erik S. Anderson, Kazuhiro Tanaka,

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Presentation on theme: "EGFR Mutation-Induced Alternative Splicing of Max Contributes to Growth of Glycolytic Tumors in Brain Cancer Ivan Babic, Erik S. Anderson, Kazuhiro Tanaka,"— Presentation transcript:

1 EGFR Mutation-Induced Alternative Splicing of Max Contributes to Growth of Glycolytic Tumors in Brain Cancer Ivan Babic, Erik S. Anderson, Kazuhiro Tanaka, Deliang Guo, Kenta Masui, Bing Li, Shaojun Zhu, Yuchao Gu, Genaro R. Villa, David Akhavan, David Nathanson, Beatrice Gini, Sergey Mareninov, Rui Li, Carolina Espindola Camacho, Siavash K. Kurdistani, Ascia Eskin, Stanley F. Nelson, William H. Yong, Webster K. Cavenee, Timothy F. Cloughesy, Heather R. Christofk, Douglas L. Black, Paul S. Mischel Cell Metabolism Volume 17, Issue 6, Pages 1000-1008 (June 2013) DOI: 10.1016/j.cmet.2013.04.013 Copyright © 2013 Elsevier Inc. Terms and Conditions Terms and Conditions

2 Figure 1 Cell Metabolism 2013 17, 1000-1008DOI: (10.1016/j.cmet.2013.04.013) Copyright © 2013 Elsevier Inc. Terms and Conditions Terms and Conditions

3 Figure 2 Cell Metabolism 2013 17, 1000-1008DOI: (10.1016/j.cmet.2013.04.013) Copyright © 2013 Elsevier Inc. Terms and Conditions Terms and Conditions

4 Figure 3 Cell Metabolism 2013 17, 1000-1008DOI: (10.1016/j.cmet.2013.04.013) Copyright © 2013 Elsevier Inc. Terms and Conditions Terms and Conditions

5 Figure 4 Cell Metabolism 2013 17, 1000-1008DOI: (10.1016/j.cmet.2013.04.013) Copyright © 2013 Elsevier Inc. Terms and Conditions Terms and Conditions

6 Cell Metabolism 2013 17, 1000-1008DOI: (10.1016/j.cmet.2013.04.013) Copyright © 2013 Elsevier Inc. Terms and Conditions Terms and Conditions

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