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Abby Erickson, RRT Review of RT 110
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Performed by: Hand Machine Available for: Short term Long term Acute care Extended home care
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S-shaped curve, relationship of plasma PO 2 and O 2 bound to Hb (SO 2 ) Flat portion: minor changes in PO 2 have little effect on SO 2 Strong Affinity! Steep portion: small drop in PO 2 causes a large drop in SO 2 Weak Affinity!
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Normal: 4-5L/min V A = Vt-V D V A = (Vt-V D )x f ↑ V A = ↓ PaCO 2, ↑ PaO 2 Hyperventilation ↓ V A = ↑ PaCO 2, ↓ PaO 2 Hypoventilation Alveolar air equation As PaCO 2 ↑ by 1mmHg, PaO 2 ↓ by 1.25mmHg...
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Degree of compensation Acid-base balance Cause: respiratory, metabolic, mixed Oxygenation – degree of hypoxemia Must interpret in the context of the clinical picture!! Requires ventilation status History, signs, symptoms Acute changes versus chronic
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P awo : zero* P bs : zero* P pl : -5cmH2O - 10cmH2O P A : +1cmH2O - 1cmH2O *unless pressure applied
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Relative ease with which a structure distends opposite of elastance Used to describe the elastic forces that oppose lung inflation V/ P = L/cmH2O 50-170ml/cmH2O normal 35/40 -100ml/cmH2O intubated patient Static Compliance Dynamic Compliance
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Frictional forces associated with ventilation Anatomic structures Tissue viscous resistance Ability of air to flow depends on Gas viscosity Gas density Length and diameter of the tube Flow rate of the gas through the tube Raw = P TA /flow cmH2O/L/sec P TA ≈ PIP – Pplat Assumes constant flow Normal 0.6-2.4 cmH2O/L/sec Intubated patients 5-7cmH2O/L/sec (and higher!)
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Attempts to mimic normal physiology Types: Iron lung – tank ventilator Chest cuirass Maintained without the need for ETT, tracheostomy, able to talk and eat Cardiovascular concerns, access to patient
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Above normal ventilating rates with below normal ventilating volumes HFPPV HFJV HFOV
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Requires airway interface Applies pressure to create gradient between mouth and lung
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ELECTRICALLY POWEREDPNEUMATICALLY POWERED Relies on electricity Wall outlet (AC), battery (DC) Powers internal motors which provide gas flow to the patient High pressure gas source Usually 2 -50psi sources, air and oxygen Built in reducing valves Pneumatic Fluidic
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Pneumatically powered – 50 psi gas sources Mixture of air and oxygen allow variable FiO2 Energy to deliver the breath Electrically powered Controls the internal function May be controlled by a microprocessor (1980’s)
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OPEN LOOPCLOSED LOOP “unintelligent” systems Does not respond to changes in patient condition Does not measure variables or change them “intelligent” systems Compares the set variable to the measured variable
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Main inspiratory line Adapter Expiratory line Expiratory valve Adjuncts Device to warm/humidify air Thermometer Nebulizer Bacteria filters
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Muscle Pressure Action of the respiratory muscles Ventilation Pressure Produced by the ventilator These pressures produce motion (flow) to deliver a volume of gas to the lung; the volume delivered depends on the lung’s characteristics
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PRESSURE CONTROLLED BREATHING VOLUME CONTROLLED BREATHING Maintains the pressure waveform in a specific pattern Pressure waveform is unaffected by changes in lung characteristics Volume and flow waveforms vary with changes in lung characteristics Maintains the volume waveform in a specific pattern Volume and flow waveforms remain unchanged Pressure waveform varies with changes in lung characteristics
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Change from exhalation to inspiration Inspiration Change from inspiration to exhalation exhalation
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Signal measured by the ventilator Begins, sustains and ends each of the four phases of the breath Trigger variable Limit variable Cycle variable
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MANDATORYSPONTANEOUS Ventilator determines start time Ventilator determines tidal volume Ventilator determines both Machine triggers and/or cycles the breath Patient determines start of breath Patient determines tidal volume delivery
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Does not require an endotracheal tube Use of NPPV has the potential: to avoid complications of intubation decrease mortality rates decrease length of stay
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Achieve exhaled tidal volume 5-7ml/kg Patient ventilator synchrony Rise time Inspiratory sensitivity Expiratory flow cycling EPAP to offset autoPEEP Oximetry Alleviating disease/disorder signs and symptoms
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Requires patient cooperation and tolerance Selection of appropriate interface Starting with low pressure initially Allow the patient to hold the mask Reassurance Requires secure fit, leaks are acceptable
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Mask discomfort Air pressures/Gas flows –gastric insufflation Aspiration pneumonia Pneumothorax Hypotension Hypoxemia, Mucus plugging Respiratory arrest
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Reversal of the cause of respiratory failure Stabilization of the patient's condition Gradually decreasing the level of support (both ventilatory and oxygenation) Gradually increase the amount of time off NPPV
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The rest of the book! Stay on top of the reading, this term moves fast Come and see me for questions, concerns and further review, I am here to help Class time is limited so plan on additional time for independent study
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