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P. 250-255 Essentials of Anatomy Seeley Stephens Tate SPECIAL SENSES PART III VISION AND THE EYE.

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Presentation on theme: "P. 250-255 Essentials of Anatomy Seeley Stephens Tate SPECIAL SENSES PART III VISION AND THE EYE."— Presentation transcript:

1 p. 250-255 Essentials of Anatomy Seeley Stephens Tate SPECIAL SENSES PART III VISION AND THE EYE

2  Eyebrows:  Perspiration, sunlight  Eyelids & lashes  Foreign objects (blink reflex)  Lubricate eyes with blinking (20/min)  Conjunctiva  Membrane inner eyelid/ covers anterior eye  Lubrication  Small amt tears  mucus ACCESSORY STRUCTURES OF THE EYE

3 LACRIMAL APPARATUS

4  Rectus muscles  Superior, inferior  Medial, lateral  Oblique muscles  Superior, inferior EXTRINSIC EYE MUSCLES

5  3 Tunics (layers)  Fibrous  Vascular  Nervous ANATOMY OF THE EYE

6  Sclera: firm white outer connective tissue  maintain shape, attachment of muscle, protect  Cornea: transparent, anterior eye  allow light to enter, refracts light FIBROUS TUNIC

7  most blood vessels  CHOROID: Thin vascular network and melanin- containing cells, appears black  CILIARY BODY: smooth muscles,  LENS: flexible transparent disc  IRIS: colored part, contractile muscles, anterior to lens, regulates diameter of the pupil  PUPIL: opening, light passes through VASCULAR TUNIC

8  Pigmented Retina: w/choroid keeps light from reflecting into the eye  Sensory Retina: contains rods/cones, interneurons  Rods: 20 x more common, sense light  Cones: need more light, sense color  6-7 million  Long (Red), Short (blue), Medium (green) varieties NERVOUS TUNIC

9  Rod cells: rhodopsin (opsin and retinal) pigment  Light changes retinal’s shape  Night blindness: vitamin A deficiency  Cone cells: blue, red, green opsin NERVOUS TUNIC

10  Rods/cones synapse with bipolar sensory cells  then ganglion (cell bodies) which converge to form optic nerve NERVOUS TUNIC

11  Fovea Centralis: only cone cells, light most focused  Macula lutea: yellow spot contains fovea centralis  Optic disc: blood vessels enter NERVOUS TUNIC  Optic disc:  blood vessels enter  optic nerve exits  “blind spot” of the eye  no photoreceptors Macula lutea Fovea Centralis

12  Anterior and Posterior chambers:  aqueous humor-watery fluid of eye, produced by ciliary body  Maintain pressure, refracts light  Vitreous chamber:  vitreous humor – jellylike substance helps maintain pressure, hold lens and retina in place CHAMBERS OF THE EYE

13 FUNCTIONS OF THE EYE

14  Cornea: Convex, light is bent so the rays converge  Lens: also convex, fine focus adjustments by changing shape  Ciliary body maintains pressure on lens, relatively flat = long distance  Object closer than 20 feet, ciliary muscles contract, ciliary body closer to lens, lens flexes more spherical  Accommodation: process of ciliary body moving lens to focus on objects closer than 20 feet ACCOMMODATION

15 EYE TESTS & DISORDERS

16  Snellen Eye Chart  At 20 feet you can read a letter that most humans can read at 20 feet  20/200 means you can read at 20 feet a letter most can read at 200 feet  LogMAR chart is considered more reliable font, spacing, number of letters STATION 1: VISUAL ACUITY

17 STATION 2: ASTIGMATISM  Misshapen cornea or lens  Blurry, fuzzy vision at all distances  Snellen Astigmatism Test  If lines appear wavy or different colors (thinner), you may have astigmatism  Clear vision, same thickness same spacing

18  Myopoia: Near Sighted (trouble with distant)  Eye focuses light in front of the retina  Long eyeball, strong lens MYOPIA & HYPEROPIA  Hyperopia:  Far Sighted  trouble close objects  5-10% of Americans  Flat lens, short eyeball

19  Decrease in ability of eye to accommodate near vision  Part of aging process  Lens is no longer flexible PRESBYOPIA

20  Optic Disc – no photoreceptors BLIND SPOT

21  August Seebeck used 300 colored papers (1837). Participants matched papers to a sample color.  Holmgren (1877) used colored wool. Colors that are difficult to distinguish can indicate color vision difficulties.  Ishichara color plates are commonly seen for color vision deficiency testing. STATION 4: COLOR VISION

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27  Can be complete or only decrease in perception  Most common cause is faulty cone development  More frequent in males (8% males with northern european ancestry, only 1% of females)  X-linked genetic trait COLOR DEFICIENCY (COLOR BLINDNESS)

28  Conjunctivitis: inflammation (usually bacterial, aka pinkeye)  Neonatal ophthalamia: severe form, high risk of blindness to infant if mother is infected  Chlamydial conjunctivitis  Gonorrheal EYE DISORDERS  Trachoma: leading infectious cause of blindness; scarring of cornea

29  A clouding in the lens  Proteins that make up the lens clump together  Most are related to aging  Smoking, diabetes, radiation also suspected causes CATARACT (b) The same scene viewed with cataracts. (a) Normal vision

30  Cataract: (22 million U.S.)  Clouding of lens due to age, infection, trauma  Glaucoma: (2 million)  Pressure build up from aqueous humor  Diabetes meliitus:  A major cause of blindness in U.S.  Damage to optic nerve, cataracts, retinal detach CAUSES OF BLINDNESS


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